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1.
Abstract

The purpose of this study was (a) to assess lactate accumulation during isometric exercise, and to quantify the shifts in accumulation following isometric training; and (b) to relate any training-induced changes in lactate accumulation to reductions in resting blood pressure. Eleven male participants undertook isometric training for a 4-week period using bilateral-leg exercise. Training caused reductions in systolic, diastolic, and mean arterial resting blood pressure (of ?4.9 ± 6.3 mmHg, P = 0.01; ?2.6 ± 3.0 mmHg, P = 0.01; and ?2.6 ± 2.3 mmHg, P = 0.001 respectively; mean ± s). These were accompanied by changes in muscle activity, taken as electromyographic activity to reach a given lactate concentration (from 114 ± 22 to 131 ± 27 mV and from 136 ± 25 to 155 ± 34 mV for 3 and 4 mmol · L?1 respectively. Training intensity expressed relative to peak lactate was correlated with reduced resting systolic and mean arterial blood pressure. Training caused significant shifts in lactate accumulation, and reductions in resting blood pressure are strongly related to training intensity, when expressed relative to pre-training peak lactate. This suggests that higher levels of local muscle anaerobiosis may promote the training-induced reductions in resting blood pressure.  相似文献   

2.
In this study, we examined the correlations between selected markers of isometric training intensity and subsequent reductions in resting blood pressure. Thirteen participants performed a discontinuous incremental isometric exercise test to volitional exhaustion at which point mean torque for the final 2-min stage (2min-torque(peak)) and peak heart rate peak (HR(peak)) were identified. Also, during 4 weeks of training (3 sessions per week, comprising 4?×?2?min bilateral leg isometric exercise at 95% HR(peak)), heart rate (HR(train)), torque (Torque(train)), and changes in EMG amplitude (ΔEMG(amp)) and frequency (ΔEMG(freq)) were determined. The markers of training intensity were: Torque(train) relative to the 2min-torque(peak) (%2min-torque(peak)), EMG relative to EMG(peak) (%EMG(peak)), HR(train) ΔEMG(amp), ΔEMG(freq), and %MVC. Mean systolic (-4.9 mmHg) and arterial blood pressure (-2.7mmHg) reductions correlated with %2min-torque(peak) (r?=?-0.65, P?=?0.02 and r?=?-0.59, P?=?0.03), ΔEMG(amp) (r?=?0.66, P?=?0.01 and r?=?0.59, P?=?0.03), ΔEMG(freq) (r?=?-0.67, P?=?0.01 and r?=?-0.64, P?=?0.02), and %EMG(peak) (systolic blood pressure only; r?=?-0.63, P?=?0.02). These markers best reflect the association between isometric training intensity and reduction in resting blood pressure observed after bilateral leg isometric exercise training.  相似文献   

3.
There is a paucity of research on devices suitable for home-based isometric exercise. Our aim was to compare cardiovascular responses to isometric exercise using novel and established methods. Ten individuals (age 34.0 +/- 8.5 years, mass 68.2 +/- 10.4 kg, height 1.72 +/- 0.09 m; mean +/- s) performed three different isometric exercise protocols with 48 h between each. Each protocol involved four repeated exercise bouts of 2 min at 30% maximum voluntary contraction force using alternate legs (transducer), alternate arms (transducer), or alternate arms (novel device). Systolic blood pressure, diastolic blood pressure, mean arterial blood pressure and heart rate were measured every 30 s. The highest (peak) values during each 2 min bout of exercise were recorded (peak systolic blood pressure, peak diastolic blood pressure, peak mean arterial blood pressure and peak heart rate). At the end of each 2 min exercise bout, the participants rated their perceived discomfort using Borg's CR-10 scale. There was a statistically significant difference in peak systolic blood pressure between isometric arm flexion using the force transducer and the novel device [158.1 +/- 10.8 vs. 149.1 +/- 13.9 mmHg (mean +/- s); P = 0.02]. Further analysis showed that peak systolic blood pressure was on average 9 mmHg higher using the force transducer with limits of agreement of - 15.97 to 33.97 mmHg. Analysis of the peak diastolic blood pressure, peak mean arterial blood pressure, peak heart rate and CR-10 data revealed no statistically significant differences between the three protocols. These results suggest that this novel, home-based method elicited similar cardiovascular responses during isometric exercise to those of established laboratory-based methods. However, the lower peak systolic blood pressure using the modified scales warrants further investigation before this method is used widely in the home.  相似文献   

4.
To reduce resting blood pressure, a minimum isometric exercise training (IET) intensity has been suggested, but this is not known for short-term IET programmes. We therefore compared the effects of moderate- and low-intensity IET programmes on resting blood pressure. Forty normotensive participants (22.3 ± 3.4 years; 69.5 ± 15.5 kg; 170.2 ± 8.7 cm) were randomly assigned to groups of differing training intensities [20%EMGpeak (~23%MVC, maximum voluntary contraction, or 30%EMGpeak (~34%MVC)] or control group; 3 weeks of IET at 30%EMGpeak resulted in significant reductions in resting mean arterial pressure (e.g. ?3.9 ± 1.0 mmHg, P < 0.001), whereas 20%EMGpeak did not (?2.3 ± 2.9 mmHg; P > 0.05). Moreover, after pooling all female versus male participants, IET induced a 6.9-mmHg reduction in systolic blood pressure in female participants, but only a 1.5-mmHg reduction in systolic blood pressure in male participants, although the difference was not significant. An IET intensity between 20%EMGpeak and 30%EMGpeak is sufficient to elicit significant resting blood pressure reductions in a short-term training period (3 weeks). In addition, sexual dimorphism may exist in the magnitude of reductions, but further work is required to confirm this possibility, which could be important in understanding the mechanisms responsible.  相似文献   

5.
There is a paucity of research on devices suitable for home-based isometric exercise. Our aim was to compare cardiovascular responses to isometric exercise using novel and established methods. Ten individuals (age 34.0?±?8.5 years, mass 68.2?±?10.4?kg, height 1.72?±?0.09?m; mean?±?s) performed three different isometric exercise protocols with 48?h between each. Each protocol involved four repeated exercise bouts of 2?min at 30% maximum voluntary contraction force using alternate legs (transducer), alternate arms (transducer), or alternate arms (novel device). Systolic blood pressure, diastolic blood pressure, mean arterial blood pressure and heart rate were measured every 30?s. The highest (peak) values during each 2?min bout of exercise were recorded (peak systolic blood pressure, peak diastolic blood pressure, peak mean arterial blood pressure and peak heart rate). At the end of each 2?min exercise bout, the participants rated their perceived discomfort using Borg's CR-10 scale. There was a statistically significant difference in peak systolic blood pressure between isometric arm flexion using the force transducer and the novel device [158.1?±?10.8 vs. 149.1?±?13.9?mmHg (mean?±?s); P = 0.02]. Further analysis showed that peak systolic blood pressure was on average 9?mmHg higher using the force transducer with limits of agreement of –?15.97 to 33.97?mmHg. Analysis of the peak diastolic blood pressure, peak mean arterial blood pressure, peak heart rate and CR-10 data revealed no statistically significant differences between the three protocols. These results suggest that this novel, home-based method elicited similar cardiovascular responses during isometric exercise to those of established laboratory-based methods. However, the lower peak systolic blood pressure using the modified scales warrants further investigation before this method is used widely in the home.  相似文献   

6.
Reduced hepatic lactate elimination initiates blood lactate accumulation during incremental exercise. In this study, we wished to determine whether renal lactate elimination contributes to the initiation of blood lactate accumulation. The renal arterial-to-venous (a-v) lactate difference was determined in nine men during sodium lactate infusion to enhance the evaluation (0.5 mol x L(-1) at 16 ± 1 mL x min(-1); mean ± s) both at rest and during cycling exercise (heart rate 139 ± 5 beats x min(-1)). The renal release of erythropoietin was used to detect kidney tissue ischaemia. At rest, the a-v O(2) (CaO(2)-CvO(2)) and lactate concentration differences were 0.8 ± 0.2 and 0.02 ± 0.02 mmol x L(-1), respectively. During exercise, arterial lactate and CaO(2)-CvO(2) increased to 7.1 ± 1.1 and 2.6 ± 0.8 mmol x L(-1), respectively (P < 0.05), indicating a -70% reduction of renal blood flow with no significant change in the renal venous erythropoietin concentration (0.8 ± 1.4 U x L(-1)). The a-v lactate concentration difference increased to 0.5 ± 0.8 mmol x L(-1), indicating similar lactate elimination as at rest. In conclusion, a -70% reduction in renal blood flow does not provoke critical renal ischaemia, and renal lactate elimination is maintained. Thus, kidney lactate elimination is unlikely to contribute to the initial blood lactate accumulation during progressive exercise.  相似文献   

7.
Currently, it is not possible to prescribe isometric exercise at an intensity that corresponds to given heart rates or systolic blood pressures. This might be useful in optimizing the effects of isometric exercise training. Therefore, the aim of this study was to explore the relationships between isometric exercise intensity and both heart rate and systolic blood pressure during repeated incremental isometric exercise tests. Fifteen participants performed seated isometric double-leg knee extension, during which maximum voluntary contraction (MVC) was assessed, using an isokinetic dynamometer. From this, a corresponding peak electromyographic activity (EMG(peak)) was determined. Subsequently, participants performed two incremental isometric exercise tests (at least 48 h apart) at 10, 15, 20, 25, and 30% EMG(peak), during which steady-state heart rate and systolic blood pressure were recorded. In all participants, there were linear relationships between %EMG(peak) and heart rate (r at least 0.91; P < 0.05) and between %EMG(peak) and systolic blood pressure (r at least 0.92; P < 0.05). Also, when repeated tests were compared, there were no differences in the slopes (P > 0.50) or elevations (P > 0.10) for either of the relationships. Therefore, these linear relationships could be used to identify isometric exercise training intensities that correspond to precise heart rates or systolic blood pressures. Training performed in this way might provide greater insight into the underlying mechanisms for the cardiovascular adaptations that are known to occur as a result.  相似文献   

8.
Abstract

In this study, we examined the correlations between selected markers of isometric training intensity and subsequent reductions in resting blood pressure. Thirteen participants performed a discontinuous incremental isometric exercise test to volitional exhaustion at which point mean torque for the final 2-min stage (2min-torquepeak) and peak heart rate peak (HRpeak) were identified. Also, during 4 weeks of training (3 sessions per week, comprising 4 × 2 min bilateral leg isometric exercise at 95% HRpeak), heart rate (HRtrain), torque (Torquetrain), and changes in EMG amplitude (ΔEMGamp) and frequency (ΔEMGfreq) were determined. The markers of training intensity were: Torquetrain relative to the 2min-torquepeak (%2min-torquepeak), EMG relative to EMGpeak (%EMGpeak), HRtrain ΔEMGamp, ΔEMGfreq, and %MVC. Mean systolic (?4.9 mmHg) and arterial blood pressure (?2.7mmHg) reductions correlated with %2min-torquepeak (r = ?0.65, P = 0.02 and r = ?0.59, P = 0.03), ΔEMGamp (r = 0.66, P = 0.01 and r = 0.59, P = 0.03), ΔEMGfreq (r = ?0.67, P = 0.01 and r = ?0.64, P = 0.02), and %EMGpeak (systolic blood pressure only; r = ?0.63, P = 0.02). These markers best reflect the association between isometric training intensity and reduction in resting blood pressure observed after bilateral leg isometric exercise training.  相似文献   

9.
In this study, we compared the effects of accumulated and continuous running on resting arterial blood pressure. Ten normotensive/pre-hypertensive men, aged 25.0 ± 4.2 years (mean ± s), participated in three 2-day trials at least one week apart in a randomized, repeated-measures design. On Day 1, participants rested (control) or ran at 70% of maximum oxygen uptake in either ten 3-min bouts (30 min rest between bouts) or one continuous 30-min bout. On Day 2, participants rested throughout the day. Blood pressure was measured at hourly intervals throughout Days 1 and 2. Mean resting systolic blood pressure on Day 2 was 6% lower during the accumulated and continuous running trials compared with the control trial (110 ± 6 vs. 110 ± 8 vs. 117 ± 6 mmHg respectively; P < 0.05), but there were no differences in resting diastolic blood pressure among the three trials (70 ± 7 vs. 69 ± 6 vs. 70 ± 5 mmHg respectively). These findings demonstrate that accumulating 30 min of running throughout the day in short bouts is as effective as 30 min of continuous running for reducing resting systolic blood pressure on the next day in young normotensive/pre-hyptertensive men.  相似文献   

10.
Individuals with impaired glucose tolerance (IGT) are at greater risk of developing diabetes than in normoglycaemia. The aim of this study was to examine the effects of 12-weeks exercise training in obese humans with IGT. Eleven participants (6 males and 5 females; 49±9 years; mean Body Mass Index (BMI) 32.4 kg · m(-2)), completed a 12-week brisk walking intervention (30 min per day, five days a week (d · wk(-1)), at 65% of age-predicted maximal heart rate (HR(max)). Anthropometric measurements, dietary intake, pulse wave velocity (PWV, to determine arterial stiffness) and blood pressure (BP) were examined at baseline and post intervention. Fasting blood glucose, glycosylated haemoglobin, insulin, blood lipids, indices of oxidative stress and inflammation (lipid hydroperoxides; superoxide dismutase; multimeric adiponectin concentration and high-sensitivity C-reactive protein) were also determined. Post intervention, PWV (9.08±1.27 m · s(-1) vs. 8.39±1.21 m · s(-1)), systolic BP (145.4±14.5 vs. 135.8±14.9 mmHg), triglycerides (1.52±0.53 mmol · L(-1) vs. 1.31±0.54 mmol · L(-1)), lipid hydroperoxides (1.20±0.47 μM · L(-1) vs. 0.79±0.32 μM · L(-1)) and anthropometric measures decreased significantly (P < 0.05). Moderate intensity exercise training improves upper limb vascular function in obese humans with IGT, possibly by improving triglyceride metabolism, which may subsequently reduce oxidative stress. These changes were independent of multimeric adiponectin modification and alterations in other blood biomarkers.  相似文献   

11.
ABSTRACT

Interval exercise training is increasingly recommended to improve health and fitness; however, it is not known if cardiovascular risk is different from continuous exercise protocols. This systematic review with meta-analyses assessed the effect of a single bout of interval exercise on cardiovascular responses that indicate risk of cardiac fibrillation and infarction compared to continuous exercise. Electronic databases Medline, CINAHL, Embase, Scopus and Cochrane were searched. Key inclusion criteria were: (1) intervals of the same intensity and duration followed by a recovery period and (2) reporting at least one of blood pressure, heart rate variability, arterial stiffness or function. Cochrane Risk of Bias tool and GRADE approach were used. Meta-analyses found that systolic blood pressure responses to interval exercise did not differ from responses to continuous exercise immediately (MD 8 mmHg [95% CI ?32, 47], p = 0.71) or at 60 min following exercise (MD 0 mmHg [95% CI ?2, 1], p = 0.79). However, reductions in diastolic blood pressure and flow-mediated dilation with interval exercise were observed 10–15 min post-exercise. The available evidence indicates that interval exercise does not convey higher cardiovascular risk than continuous exercise. Further investigation is required to establish the safety of interval exercise for clinical populations.  相似文献   

12.
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13.
We tested the hypothesis that exercise-induced muscle damage would increase the ventilatory (V(E)) response to incremental/ramp cycle exercise (lower the gas exchange threshold) without altering the blood lactate profile, thereby dissociating the gas exchange and lactate thresholds. Ten physically active men completed maximal incremental cycle tests before (pre) and 48 h after (post) performing eccentric exercise comprising 100 squats. Pulmonary gas exchange was measured breath-by-breath and fingertip blood sampled at 1-min intervals for determination of blood lactate concentration. The gas exchange threshold occurred at a lower work rate (pre: 136 ± 27 W; post: 105 ± 19 W; P < 0.05) and oxygen uptake (VO(2)) (pre: 1.58 ± 0.26 litres · min(-1); post: 1.41 ± 0.14 litres · min(-1); P < 0.05) after eccentric exercise. However, the lactate threshold occurred at a similar work rate (pre: 161 ± 19 W; post: 158 ± 22 W; P > 0.05) and VO(2) (pre: 1.90 ± 0.20 litres · min(-1); post: 1.88 ± 0.15 litres · min(-1); P > 0.05) after eccentric exercise. These findings demonstrate that exercise-induced muscle damage dissociates the V(E) response to incremental/ramp exercise from the blood lactate response, indicating that V(E) may be controlled by additional or altered neurogenic stimuli following eccentric exercise. Thus, due consideration of prior eccentric exercise should be made when using the gas exchange threshold to provide a non-invasive estimation of the lactate threshold.  相似文献   

14.
有氧运动对轻度原发性高血压的疗效及内皮功能的影响   总被引：1，自引：0，他引：1  
目的：观察有氧运动对轻度原发性高血压患者的疗效和内皮功能的影响。方法：40例轻度原发性局血压患者进行3个月的低强度有氧运动，用超声测定肱动脉内皮依赖性血管舒张功能（FMD）和非内皮依赖性血管舒张-即含服硝酸甘油时血管舒张功能（NTG—D），记录并比较运动前、后的血压、心率及FMD、NTG—D，并与36例健康体检者对照比较。结果：运动前轻度原发性高血压患者FMD明显下降，与对照组相比，P〈0．01，肱动脉基础内径和NTG～D差异无统计学意义（P〉0．05）；运动后轻度原发性高血压患者血压从（150．2±8．5）／（85．3±7．3）mmHg下降至（137．2±7．6）／（77．3±7．0）mmHg（P〈0．05），同时治疗后FMD较治疗前显著增加（P〈0．01）．但是与对照组比较差异仍有统计学意义（P〈0．05），运动前、后NTG—D无变化（P〉0．05）。结论：低强度有氧运动可降低轻度原发性高血压患者的血压，提高FMD，改善内皮功能。  相似文献   

15.
刘弢 《中国体育科技》2007,43(5):129-131
目的:考察11岁左右少年运动员开始进行大运动量训练的3个项目(自行车、划船、足球)的动脉血压反应,并分析最大运动时动脉血压、VO_2max对受试者心脏体积增加的影响。方法:对受试者进行病史和体格检查,用M型二堆超声心动图、12导心电图检查心脏参数,逐级运动试验直接测定VO_2max。在安静和最大运动状态下检测收缩压和舒张压。用Devereux公式计算左心室体积指数(LVMI)。结果:自行车运动员的LVMI值明显高于其他运动员;划船运动员在最大运动时的动脉血压最高,而自行车运动员具有较高的VO_2max。LVMI与VO_2max和最大运动时的收缩压有较好的相关。结论:VO_2max对运动员心脏肥大的发生和发展起重要作用。较大的动脉血压反应有利于发展较大的LVMI。  相似文献   

16.
The effect of inspiratory muscle training for 10 min twice a day for 27.5 days was evaluated in 20 human subjects, of whom 10 formed a training group and 10 a sham training group. The maximal oxygen uptake (VO2 max), maximal ventilation, breathing frequency during maximal exercise and the distance run in 12 min on a track were determined in addition to resting peak expiratory flow, forced vital capacity (FVC) and forced expiratory volume in 1 s (FEV1), with alveolar oxygen tension (pAO2) during maximal exercise being calculated. Inspiratory muscle training increased maximal inspiratory pressure from 93 (range 38-118) to 110 (65-165) mmHg in the training group (P less than 0.0005), but did not affect VO2 max, ventilation during maximal exercise, peak expiratory flow, FEV1 or FVC. However, breathing frequency during maximal exercise decreased slightly from 56 (44-87) to 53 (38-84) breaths min-1 (P less than 0.05) in the training group only; but the calculated pAO2 did not increase from the pre-training value of 126 (116-132) mmHg. The maximal distance run during 12 min increased similarly in the training and sham training groups by 8% (3-12%) and 6% (2-12%), respectively (P less than 0.01). The results of this study show that inspiratory muscle training resulting in a 32% (0-85%) increase in maximal inspiratory pressure does not change FEV1, FVC, peak expiratory flow, VO2 max or work capacity.  相似文献   

17.
We test the hypothesis that breathing oxygen-enriched air (F(I)O(2) = 100%) maintains exercise performance and reduces fatigue during intervals of maximal-intensity cycling. Ten well-trained male cyclists (age 25 ± 3 years; peak oxygen uptake 64.8 ± 6.2 ml · kg(-1) · min(-1); mean ± s) were exposed to either hyperoxic or normoxic air during the 6-min intervals between five 30-s sessions of cycling at maximal intensity. The concentrations of lactate and hydrogen ions [H(+)], pH, base excess, oxygen partial pressure, and oxygen saturation in the blood were assessed before and after these sprints. The peak (P = 0.62) and mean power outputs (P = 0.83) with hyperoxic and normoxic air did not differ. The partial pressure of oxygen was 4.2-fold higher after inhaling hyperoxic air, whereas lactate concentration, pH, [H(+)], and base excess (P ≥ 0.17) were not influenced. Perceived exertion towards the end of the 6-min periods after the fourth and fifth sprints (P < 0.05) was lower with hyperoxia than normoxia (P < 0.05). These findings demonstrate that the peak and mean power outputs of athletes performing intervals of maximal-intensity cycling are not improved by inhalation of oxygen-enriched air during recovery.  相似文献   

18.
本实验通过测定全国甲级Ａ组足球运动员，在功率自行车上运动时及恢复期间心率、收缩压、舒张压、脉压差及平均动脉压的变化，发现心率、收缩压在负荷时适度平行升高，舒张压在负荷时适度下降（１２ｍｍＨｇ）．脉压差负荷时逐渐加大，平均动脉压负荷时稍有上升（１１ｍｍＨｇ），恢复期最为稳定，各项指标均在６—１２ｍｉｎ内恢复到安静水平。说明其运动潜力还很大，并指出应加强综合心功的能力训练来适应高强度训练中肌肉运动的需要。  相似文献   

19.
Twelve endurance athletes and six power athletes performed fatiguing isokinetic knee flexions/extensions. Isokinetic torque was recorded during the exercise. Isometric torque, cortisol and lactate responses, electromyographic (EMG) mean power frequency, average rectified value, and conduction velocity were analysed before and after the isokinetic exercise to determine correlations between electrophysiological variables and mechanical performances and/or blood concentrations of biomarkers in the two groups of athletes. The EMG variables were estimated from signals recorded from the vastus lateralis in both voluntary and electrically elicited isometric contractions. Power athletes recorded higher values than endurance athletes for the following variables: pre-exercise isometric maximal voluntary contraction (MVC), isokinetic MVC, rate of mechanical fatigue during isokinetic contractions, pre - post exercise variations and recovery times of conduction velocity and mean power frequency, and lactate concentrations. Moreover, conduction velocity overshooting was observed in endurance athletes during the recovery phase after exercise. The correlation analyses showed that the higher the rate of mechanical fatigue, the higher the lactate production and the reduction in conduction velocity due to the exercise.  相似文献   

20.
We have previously shown that single-leg training results in improved endurance for exercise with the untrained leg (UTL) as well as for exercise with the trained leg (TL). The purpose of this study was to see whether the improved endurance of the untrained leg could be explained on the basis of changes in muscle metabolism. Exercise time to exhaustion at 80% of maximum oxygen uptake (VO2 max) was determined for each leg separately, pre- and post-training. Muscle metabolite concentrations were measured pre- and post-training in biopsy samples obtained immediately before this endurance test and at the pre-training point of exhaustion (END1). After six weeks of single-leg training endurance time was increased for both the UTL and the TL (UTL 34.0 +/- 16.4 min vs 97.9 +/- 26.3 min, P less than 0.01; TL 28.3 +/- 10.1 min vs 169.0 +/- 32.6 min, P less than 0.01). No changes in muscle metabolite concentrations were found in resting muscle. Training increased muscle ATP (P less than 0.05) and glycogen (P less than 0.01) concentrations and decreased muscle lactate concentration (P less than 0.05) in the TL at END1. No significant changes in muscle metabolite concentrations were found for the UTL. The improved endurance of the contralateral limb after single-leg training could not be explained on the basis of changes in muscle metabolism.  相似文献   

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