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1.
There are few data in the published literature on sweat loss and drinking behaviour in athletes training in a cool environment. Sweat loss and fluid intake were measured in 17 first-team members of an elite soccer team training for 90 min in a cool (5 degrees C, 81% relative humidity) environment. Sweat loss was assessed from the change in body mass after correction for the volume of fluid consumed. Sweat electrolyte content was measured from absorbent patches applied at four skin sites. Mean (+/- s) sweat loss during training was 1.69+/-0.45 l (range 1.06-2.65 l). Mean fluid intake during training was 423+/-215 ml (44-951 ml). There was no apparent relationship between the amount of sweat lost and the volume of fluid consumed during training (r2 = 0.013, P = 0.665). Mean sweat sodium concentration was 42.5+/-13.0 mmol l(-1) and mean sweat potassium concentration was 4.2+/-1.0 mmol x l(-1). Total salt (NaCl) loss during training was 4.3+/-1.8 g. The sweat loss data are similar to those recorded in elite players undergoing a similar training session in warm environments, but the volume of fluid ingested is less.  相似文献   

2.
Observational research on professional athletes from the USA suggests differences may exist in sweat sodium loss based on ethnic differences. The New Zealand (NZ) sporting population is mainly of European or Māori/Pacific Island origin. Therefore, this study aimed to describe the fluid-electrolyte balance of athletes by ethnicity. A total of 20 Māori/Pacific Islanders (MP; body mass 100.97 ± 13.05 kg) and 29 NZ European (NZE; body mass 89.11 ± 11.56 kg) elite male athletes were recruited. Sweat rates were determined by body mass change during a 1-h spin cycle exercise session, during which fluid intakes and heart rate were recorded. Sweat samples were analysed for sodium concentration. Mean ± SD sweat sodium concentrations were 73.4 ± 27.2 mmol·L?1 and 55.5 ± 26.8 mmol·L?1 for the MP and NZE groups, respectively (p = 0.070). Sweat rate was 0.93 ± 0.26 L·h?1 for the MP group and 0.89 ± 0.33 L·h?1 for the NZE group (p = 0.357). Fluid intake was 1.05 ± 0.48 L and 0.93 ± 0.49 L for MP and NZE, respectively (p = 0.395). Half of the MP group gained weight during the exercise session compared to 37% of the NZE group. Pre-exercise urine specific gravity was significantly lower amongst the NZE group (1.016 ± 0.009 g mL?1) than the MP group (1.024 ± 0.008 g mL?1) p = 0.001. There was no significant difference in heart rate between the groups, p = 0.082. Hydration practices of athletes in NZ may differ by ethnicity, and this may highlight the need for more targeted education by ethnicity.  相似文献   

3.
Background: To determine athletes perceived and measured indices of fluid balance during training and the influence of hydration strategy use on these parameters. Methods: Thirty-three professional rugby union players completed a 120 minute training session in hot conditions (35°C, 40% relative humidity). Pre-training hydration status, sweat loss, fluid intake and changes in body mass (BM) were obtained. The use of hydration assessment techniques and players perceptions of fluid intake and sweat loss were obtained via a questionnaire. Results: The majority of players (78%) used urine colour to determine pre-training hydration status but the use of hydration assessment techniques did not influence pre-training hydration status (1.025?±?0.005 vs. 1.023?±?0.013?g.ml?1, P?=?.811). Players underestimated sweat loss (73?±?17%) to a greater extent than fluid intake (37?±?28%) which resulted in players perceiving they were in positive fluid balance (0.5?±?0.8% BM) rather than the measured negative fluid balance (?1.0?±?0.7% BM). Forty-eight percent of players used hydration monitoring strategies during exercise but no player used changes in BM to help guide fluid replacement. Conclusion: Players have difficulty perceiving fluid intake and sweat loss during training. However, the use of hydration monitoring techniques did not affect fluid balance before or during training.  相似文献   

4.
There is limited research studying fluid and electrolyte balance in rugby union players, and a paucity of information regarding the test–retest reliability. This study describes the fluid balance of elite rugby union players across multiple squads and the reliability of fluid balance measures between two equivalent training sessions. Sixty-one elite rugby players completed a single fluid balance testing session during a game simulation training session. A subsample of 21 players completed a second fluid balance testing session during an equivalent training session. Players were weighed in minimal clothing before and after each training session. Each player was provided with their own drinks which were weighed before and after each training session. More players gained body weight (9 (14.8%)) during training than lost greater than 2% of their initial body mass (1 (1.6%)). Pre-training body mass and rate of fluid loss were significantly associated (r?=?0.318, p?=?.013). There was a significant correlation between rate of fluid loss in sessions 1 (1.74?±?0.32?L?h?1) and 2 (1.10?±?0.31?L.?h?1), (r?=?0.470, p?=?.032). This could be useful for nutritionists working with rugby squads to identify players with high sweat losses.  相似文献   

5.
Abstract

Aspects of team players' performance are negatively affected when ~ 2% body mass is lost by perspiration. Although such dehydration is likely reached during summer practice in outdoors sports, it is unclear if such dehydration is achieved during the practice of indoor sports. We assessed the fluid and electrolyte deficits of elite team players during practice for the following indoor sports: indoor soccer (n=9), basketball (n=11), volleyball (n=10), and handball (n=13). Morning hydration status was estimated by measuring urine specific gravity. Sweat rate was calculated from body mass changes and fluid intake. Sweat sodium concentration from the forearm was used to estimate whole-body sodium losses. Over 91% of the players were moderately hypohydrated (urine specific gravity>1.020) at waking 3 h before practice. Indoor soccer players sweated at a higher rate (1.8 litres · h?1) than volleyball and handball players (1.2 and 1.1 litres · h?1, respectively; P<0.05), whereas sweat rate was not different between basketball players (1.5 litres · h?1) and the other team sport players (P>0.05). In average, 62±13% of sweat losses were replaced and teams' body mass loss did not exceed 1.2±0.3%. Sodium losses were similar among teams, averaging 1.2±0.2 g. The exercise fluid replacement habits of professional indoor team players are adequate to prevent 2% dehydration. However, most players could benefit from increasing fluid intake between workouts to offset the high prevalence of morning hypohydration.  相似文献   

6.
Abstract

In this study we investigated pre-training hydration status, fluid intake, and sweat loss in 20 elite male Brazilian adolescent soccer players (mean ± s: age 17.2 ± 0.5 years; height 1.76 ± 0.05 m; body mass 69.9 ± 6.0 kg) on three consecutive days of typical training during the qualifying phase of the national soccer league. Urine specific gravity (USG) and body mass changes were evaluated before and after training sessions to estimate hydration status. Players began the days of training mildly hypohydrated (USG > 1.020) and fluid intake did not match fluid losses. It was warmer on Day 1 (33.1 ± 2.4°C and43.4 ± 3.2% relative humidity; P < 0.05) and total estimated sweat losses (2822 ± 530 mL) and fluid intake (1607 ± 460 mL) were significantly higher (P < 0.001) compared with Days 2 and 3. Data also indicate a significant correlation between the extent of sweat loss and the volume of fluid consumed (Day 1: r = 0.560, P = 0.010; Day 2: r = 0.445, P = 0.049; Day 3: r = 0.743, P = 0.0001). We conclude that young, native tropical soccer players arrive hypohydrated to training and that they exhibit voluntary dehydration; therefore, enhancing athletes' self-knowledge of sweat loss during training might help them to consume sufficient fluid to match the sweat losses.  相似文献   

7.
Sweat lactate reflects eccrine gland metabolism. However, the metabolic tendencies of eccrine glands in a hot versus thermoneutral environment are not well understood. Sixteen male volunteers completed a maximal cycling trial and two 60-min cycling trials [30°C?=?30±1°C and 18°C?=?18±1°C wet bulb globe temperature (WBGT)]. The participants were requested to maintain a cadence of 60 rev?·?min?1 with the intensity individualized at ～ 90% of the ventilatory threshold. Sweat samples at 10, 20, 30, 40, 50 and 60?min were analysed for lactate concentration. Sweat rate at 30°C (1380±325?ml?·?h?1) was significantly greater (P<0.05) than at 18°C (632±311?ml?·?h?1). Sweat lactate concentration was significantly greater (P<0.05) at each time point during the 18°C trial, with values between trials tending to converge across time. During the 30°C trial, both heart rate (20, 30, 40, 50 and 60?min) and rectal temperature (30, 40, 50 and 60?min) were significantly higher than in the 18°C trial. Higher sweat lactate concentrations coupled with lower sweat rates may indicate a greater relative contribution of oxygen-independent metabolism within eccrine glands during exercise at 18°C. Decreases in sweat lactate concentration across time suggest either greater dilution due to greater sweat volume or increased reliance on aerobic metabolism within eccrine glands. The convergence of lactate concentrations between trials may indicate that time-dependent modifications in sweat gland metabolism occur at different rates contingent partially on environmental conditions.  相似文献   

8.
Nine male student games players consumed either flavoured water (0.1 g carbohydrate, Na+ 6 mmol · l?1), a solution containing 6.5% carbohydrate-electrolytes (6.5 g carbohydrate, Na+ 21 mmol · l?1) or a taste placebo (Na+ 2 mmol · l?1) during an intermittent shuttle test performed on three separate occasions at an ambient temperature of 30°C (dry bulb). The test involved five 15-min sets of repeated cycles of walking and variable speed running, each separated by a 4-min rest (part A of the test), followed by 60 s run/60 s rest until exhaustion (part B of the test). The participants drank 6.5 ml · kg?1 of fluid as a bolus just before exercise and thereafter 4.5 ml · kg?1 during every exercise set and rest period (19 min). There was a trial order effect. The total distance completed by the participants was greater in trial 3 (8441 ± 873 m) than in trial 1 (6839 ± 512, P < 0.05). This represented a 19% improvement in exercise capacity. However, the trials were performed in a random counterbalanced order and the participants completed 8634 ± 653 m, 7786 ± 741 m and 7099 ± 647 m in the flavoured water (FW), placebo (P) and carbohydrate-electrolyte (CE) trials, respectively (P = 0.08). Sprint performance was not different between the trials but was impaired over time (FW vs P vs CE: set 1, 2.41 ± 0.02 vs 2.39 ± 0.03 vs 2.39 ± 0.03 s; end set, 2.46 ± 0.03 vs 2.47 ± 0.03 vs 2.47 ± 0.02 s; main

effect time, P < 0.01). The rate of rise in rectal temperature was greater in the carbohydrate-electrolyte trial (rise in rectal temperature/duration of trial, °C · h?1; FW vs CE, P < 0.05; P vs CE, N.S.). Blood glucose concentrations were higher in the carbohydrate-electrolyte than in the other two trials (FW vs P vs CE: rest, 4.4 ± 0.1 vs 4.3 ± 0.1 vs 4.2 ± 0.1 mmol · l?1; end of exercise, 5.4 ± 0.3 vs 6.4 ± 0.6 vs 7.2 ± 0.5 mmol · l?1; main effect trial, P < 0.05; main effect time, P < 0.01). Plasma free fatty acid concentrations at the end of exercise were lower in the carbohydrate-electrolyte trial than in the other two trials (FW vs P vs CE: 0.57 ± 0.08 vs 0.53 ± 0.11 vs 0.29 ± 0.04 mmol · l?1; interaction, P < 0.01). The correlation between the rate of rise in rectal temperature (°C · h?1) and the distance completed was ?0.91, ?0.92 and ?0.96 in the flavoured water, placebo and carbohydrate-electrolyte conditions, respectively (P < 0.01). Heart rate, blood pressure, plasma ammonia, blood lactate, plasma volume and rate of perceived exertion were not different between the three fluid trials. Although drinking the carbohydrate-electrolyte solution induced greater metabolic changes than the flavoured water and placebo solutions, it is unlikely that in these unacclimated males carbohydrate availability was a limiting factor in the performance of intermittent running in hot environmental conditions.  相似文献   

9.
Abstract

The aim of this study was to determine the effect of 12 weeks of training on the critical velocity and maximal lactate steady state of elite swimmers. The tests to determine critical velocity and maximal lactate steady state were performed before and after 12 weeks of training. Critical velocity after 12 weeks of training was significantly higher than before training (1.45±0.10 m · s?1 vs. 1.41±0.11 m · s?1). In contrast, no significant differences in the velocity at maximal lactate steady state were observed before and after training (1.41±0.10 m · s?1 vs. 1.43±0.10 m · s?1). There was also a decrease in mean lactate concentration after 12 weeks of training. Before training, the velocity at maximal lactate steady state occurred at 100% of critical velocity, with a mean lactate concentration of 4.34 mmol · l?1. After training, the velocity at maximal lactate steady state occurred at 98% of critical velocity, with a reduced mean lactate concentration of 3.69 mmol · l?1. Based on these results, it would appear that 12 weeks of training was enough to promote an increase in critical velocity. Although no significant differences in the velocity at maximal lactate steady state were observed before and after training, the decrease in mean lactate concentration after training demonstrated greater efficiency of the aerobic system, leading to less wear during the tests.  相似文献   

10.
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11.
The purpose of this study was to examine the influence of a carbohydrate-rich meal on post-prandial metabolic responses and skeletal muscle glycogen concentration. After an overnight fast, eight male recreational/club endurance runners ingested a carbohydrate (CHO) meal (2.5 g CHO?·?kg?1 body mass) and biopsies were obtained from the vastus lateralis muscle before and 3 h after the meal. Ingestion of the meal resulted in a 10.6?±?2.5% (P?<?0.05) increase in muscle glycogen concentration (pre-meal vs post-meal: 314.0?±?33.9 vs 347.3?±?31.3 mmol?·?kg?1 dry weight). Three hours after ingestion, mean serum insulin concentrations had not returned to pre-feeding values (0 min vs 180 min: 45?±?4 vs 143?±?21 pmol?·?l?1). On a separate occasion, six similar individuals ingested the meal or fasted for a further 3 h during which time expired air samples were collected to estimate the amount of carbohydrate oxidized over the 3 h post-prandial period. It was estimated that about 20% of the carbohydrate consumed was converted into muscle glycogen, and about 12 % was oxidized. We conclude that a meal providing 2.5 g CHO?·?kg?1 body mass can increase muscle glycogen stores 3 h after ingestion. However, an estimated 67% of the carbohydrate ingested was unaccounted for and this may have been stored as liver glycogen and/or still be in the gastrointestinal tract.  相似文献   

12.
Aim: The aim of this study was to examine the relationship between ventilatory adaptation and performance during altitude training at 2700?m. Methods: Seven elite cyclists (age: 21.2?±?1.1?yr, body mass: 69.9?±?5.6?kg, height 176.3?±?4.9?cm) participated in this study. A hypoxic ventilatory response (HVR) test and a submaximal exercise test were performed at sea level prior to the training camp and again after 15 d at altitude (ALT15). Ventilation (VE), end-tidal carbon-dioxide partial pressure (PETCO2) and oxyhaemoglobin saturation via pulse oximetry (SpO2) were measured at rest and during submaximal cycling at 250?W. A hill climb (HC) performance test was conducted at sea level and after 14 d at altitude (ALT14) using a road of similar length (5.5–6?km) and gradient (4.8–5.3%). Power output was measured using SRM cranks. Average HC power at ALT14 was normalised to sea level power (HC%). Multiple regression was used to identify significant predictors of performance at altitude. Results: At ALT15, there was a significant increase in resting VE (10.3?±?1.9 vs. 12.2?±?2.4?L·min?1) and HVR (0.34?±?0.24 vs. 0.71?±?0.49?L·min?1·%?1), while PETCO2 (38.4?±?2.3 vs. 32.1?±?3.3?mmHg) and SpO2 (97.9?±?0.7 vs. 94.0?±?1.7%) were reduced (P?VE at altitude as significant predictors of HC% (adjusted r2?=?0.913; P?=?0.003). Conclusions: Ventilatory acclimatisation occurred during a 2 wk altitude training camp in elite cyclists and a higher HVR was associated with better performance at altitude, relative to sea level. These results suggest that ventilatory acclimatisation is beneficial for cycling performance at altitude.  相似文献   

13.
In this study we investigated pre-training hydration status, fluid intake, and sweat loss in 20 elite male Brazilian adolescent soccer players (mean?±?s: age 17.2?±?0.5 years; height 1.76?±?0.05?m; body mass 69.9?±?6.0?kg) on three consecutive days of typical training during the qualifying phase of the national soccer league. Urine specific gravity (USG) and body mass changes were evaluated before and after training sessions to estimate hydration status. Players began the days of training mildly hypohydrated (USG?>?1.020) and fluid intake did not match fluid losses. It was warmer on Day 1 (33.1?±?2.4°C and43.4?±?3.2% relative humidity; P?相似文献   

14.
The aims of this study were: (1) to identify the exercise intensity that corresponds to the maximal lactate steady state in adolescent endurance-trained runners; (2) to identify any differences between the sexes; and (3) to compare the maximal lactate steady state with commonly cited fixed blood lactate reference parameters. Sixteen boys and nine girls volunteered to participate in the study. They were first tested using a stepwise incremental treadmill protocol to establish the blood lactate profile and peak oxygen uptake ([Vdot]O2). Running speeds corresponding to fixed whole blood lactate concentrations of 2.0, 2.5 and 4.0?mmol?·?l?1 were calculated using linear interpolation. The maximal lactate steady state was determined from four separate 20-min constant-speed treadmill runs. The maximal lactate steady state was defined as the fastest running speed, to the nearest 0.5?km?·?h?1, where the change in blood lactate concentration between 10 and 20?min was?<0.5?mmol?·?l?1. Although the boys had to run faster than the girls to elicit the maximal lactate steady state (15.7 vs 14.3?km?·?h?1, P?<0.01), once the data were expressed relative to percent peak [Vdot]O2 (85 and 85%, respectively) and percent peak heart rate (92 and 94%, respectively), there were no differences between the sexes (P?>0.05). The running speed and percent peak [Vdot]O2 at the maximal lactate steady state were not different to those corresponding to the fixed blood lactate concentrations of 2.0 and 2.5?mmol?·?l?1 (P?>0.05), but were both lower than those at the 4.0?mmol?·?l?1 concentration (P?<0.05). In conclusion, the maximal lactate steady state corresponded to a similar relative exercise intensity as that reported in adult athletes. The running speed, percent peak [Vdot]O2 and percent peak heart rate at the maximal lactate steady state are approximated by the fixed blood lactate concentration of 2.5?mmol?·?l?1 measured during an incremental treadmill test in boys and girls.  相似文献   

15.
This study examined the effects of different work?–?rest durations during 40?min intermittent treadmill exercise and subsequent running performance. Eight males (mean?±?s: age 24.3?±?2.0 years, body mass 79.4?±?7.0?kg, height 1.77?±?0.05?m) undertook intermittent exercise involving repeated sprints at 120% of the speed at which maximal oxygen uptake (v-[Vdot]O2max) was attained with passive recovery between each one. The work?–?rest ratio was constant at 1:1.5 with trials involving short (6:9?s), medium (12:18?s) or long (24:36?s) work?–?rest durations. Each trial was followed by a performance run to volitional exhaustion at 150% v-[Vdot]O2max. After 40?min, mean exercise intensity was greater during the long (68.4?±?9.3%) than the short work?–?rest trial (54.9?±?8.1% [Vdot]O2max; P?<?0.05). Blood lactate concentration at 10?min was higher in the long and medium than in the short work?–?rest trial (6.1?±?0.8, 5.2?±?0.9, 4.5?±?1.3?mmol?·?l?1, respectively; P?<?0.05). The respiratory exchange ratio was consistently higher during the long than during the medium and short work?–?rest trials (P <?0.05). Plasma glucose concentration was higher in the long and medium than in the short work?–?rest trial after 40?min of exercise (5.6?±?0.1, 6.6?±?0.2 and 5.3?±?0.5?mmol?·?l?1, respectively; P?<?0.05). No differences were observed between trials for performance time (72.7?±?14.9, 63.2?±?13.2, 57.6?±?13.5?s for the short, medium and long work?–?rest trial, respectively; P = 0.17), although a relationship between performance time and 40?min plasma glucose was observed (P?<?0.05). The results show that 40?min of intermittent exercise involving long and medium work?–?rest durations elicits greater physiological strain and carbohydrate utilization than the same amount of intermittent exercise undertaken with a short work?–?rest duration.  相似文献   

16.
The addition of whey protein to a carbohydrate–electrolyte drink has been shown to enhance post-exercise rehydration when a volume below that recommended for full fluid balance restoration is provided. We investigated if this held true when volumes sufficient to restore fluid balance were consumed and if differences might be explained by changes in plasma albumin content. Sixteen participants lost ~1.9% of their pre-exercise body mass by cycling in the heat and rehydrated with 150% of body mass lost with either a 60 g · L?1 carbohydrate drink (CHO) or a 60 g · L?1 carbohydrate, 20 g · L?1 whey protein isolate drink (CHO-P). Urine and blood samples were collected pre-exercise, post-exercise, post-rehydration and every hour for 4 h post-rehydration. There was no difference between trials for total urine production (CHO 1057 ± 319 mL; CHO-P 970 ± 334 mL; P = 0.209), drink retention (CHO 51 ± 12%; CHO-P 55 ± 15%; P = 0.195) or net fluid balance (CHO ?393 ± 272 mL; CHO-P ?307 ± 331 mL; P = 0.284). Plasma albumin content relative to pre-exercise was increased from 2 to 4 h during CHO-P only. These results demonstrate that the addition of whey protein isolate to a carbohydrate–electrolyte drink neither enhances nor inhibits rehydration. Therefore, where post-exercise protein ingestion might benefit recovery, this can be consumed without effecting rehydration.  相似文献   

17.
This study investigated the effect of a single session of resistance exercise on postprandial lipaemia. Eleven healthy normolipidaemic men with a mean age of 23 (standard error = 1.4) years performed two trials at least 1 week apart in a counterbalanced randomized design. In each trial, participants consumed a test meal (1.2?g fat, 1.1?g carbohydrate, 0.2?g protein and 68 kJ?·?kg?1 body mass) between 08.00 and 09.00?h following a 12?h fast. The afternoon before one trial, the participants performed an 88?min bout of resistance exercise. Before the other trial, the participants were inactive (control trial). Resistance exercise was performed using free weights and included four sets of 10 repetitions of each of 11 exercises. Sets were performed at 80% of 10-repetition maximum with a 2?min work and rest interval. Venous blood samples were obtained in the fasted state and at intervals for 6?h postprandially. Fasting plasma triacylglycerol (TAG) concentration did not differ significantly between control (1.03?±?0.13?mmol?·?l?1) and exercise (0.94?±?0.09?mmol?·?l?1) trials (mean?± standard error). Similarly, the 6?h total area under the plasma TAG concentration versus time curve did not differ significantly between the control (9.84?±?1.40?mmol?·?l?1?·?6?h?1) and exercise (9.38?±?1.12?mmol?·?l?1?·?6?h?1) trials. These findings suggest that a single session of resistance exercise does not reduce postprandial lipaemia.  相似文献   

18.
Strenuous physical exercise of the limb muscles commonly results in damage, especially when that exercise is intense, prolonged and includes eccentric contractions. Many factors contribute to exercise-induced muscle injury and the mechanism is likely to differ with the type of exercise. Competitive sports players are highly susceptible to this type of injury. AM3 is an orally administered immunomodulator that reduces the synthesis of proinflammatory cytokines and normalizes defective cellular immune fractions. The ability of AM3 to prevent chronic muscle injury following strenuous exercise characterized by eccentric muscle contraction was evaluated in a double-blind and randomized pilot study. Fourteen professional male volleyball players from the First Division of the Spanish Volleyball League volunteered to take part. The participants were randomized to receive either placebo (n?=?7) or AM3 (n?=?7). The physical characteristics (mean±s) of the placebo group were as follows: age 25.7±2.1 years, body mass 87.2±4.1?kg, height 1.89±0.07?m, maximal oxygen uptake 65.3±4.2?ml?·?kg?1?·?min?1. Those of the AM3 group were as follows: age 26.1±1.9 years, body mass 85.8±6.1?kg, height 1.91±0.07?m, maximal oxygen uptake 64.6±4.5?ml?·?kg?1?·?min?1. All participants were evaluated for biochemical indices of muscle damage, including concentrations of aspartate aminotransferase, alanine aminotransferase, alkaline phosphatase, creatine kinase (CK) and its MB fraction (CK-MB), myoglobin, lactate dehydrogenase, urea, creatinine and γ-glutamyltranspeptidase, both before and 30 days after treatment (over the peak of the competitive season). In the placebo group, competitive exercise (i.e. volleyball) was accompanied by significant increases in creatine kinase (494±51 to 560±53?IU?·?l?1, P?<?0.05) and myoglobin (76.8±2.9 to 83.9±3.1?μg?·?l?1, P?<?0.05); aspartate aminotransferase (30.8±3.0 to 31.1±2.9?IU?·?l?1) and lactate dehydrogenase (380±31 to 376±29?IU?·?l?1) were relatively unchanged after the 30 days maximum effort. AM3 not only inhibited these changes, it led to a decrease from baseline serum concentrations of creatine kinase (503±49 to 316±37?IU?·?l?1, P?<?0.05) and myoglobin (80.1±3.2 to 44.1±2.6?IU?·?l?1, P?<?0.05), as well as aspartate aminotransferase (31.1±3.3 to 26.1±2.7?IU?·?l?1, P?<?0.05) and lactate dehydrogenase (368±34 to 310±3?IU?·?l?1, P?<?0.05). The concentration of CK-MB was also significantly decreased from baseline with AM3 treatment (11.6±1.2 to 5.0±0.7?IU?·?l?1, P?<?0.05), but not with placebo (11.4±1.1 to 10.8±1.4?IU?·?l?1). In conclusion, the use of immunomodulators, such as AM3, by elite sportspersons during competition significantly reduces serum concentrations of proteins associated with muscle damage.  相似文献   

19.
The thermoregulatory responses of upper-body trained athletes were examined at rest, during prolonged arm crank exercise and recovery in cool (21.5 ± 0.9°C, 43.9 ± 10.1% relative humidity; mean ± s) and warm (31.5 &± 0.6°C, 48.9 - 8.4% relative humidity) conditions. Aural temperature increased from rest by 0.7 ± 0.7°C (P ? 0.05) during exercise in cool conditions and by 1.6 ± 0.7°C during exercise in warm conditions (P ? 0.05). During exercise in cool conditions, calf skin temperature decreased (1.5 ± 1.3°C), whereas an increase was observed during exercise in warm conditions (3.0 ± 1.7°C). Lower-body skin temperatures tended to increase by greater amounts than upper-body skin temperatures during exercise in warm conditions. No differences were observed in blood lactate, heart rate or respiratory exchange ratio responses between conditions. Perceived exertion at 45 min of exercise was greater than that reported at 5 min of exercise during the cool trial (P ? 0.05), whereas during exercise in the warm trial the rating of perceived exertion increased from initial values by 30 min (P ? 0.05). Heat storage, body mass losses and fluid consumption were greater during exercise in warm conditions (7.06 ± 2.25 J·g-1 ·°C-1, 1.3 ± 0.5 kg and 1038 ± 356 ml, respectively) than in cool conditions (1.35 ± 0.23 J·g-1·°C-1, 0.8 ± 0.2 kg and 530 ± 284 ml, respectively; P ? 0.05). The results of this study indicate that the increasing thermal strain with constant thermal stress in warm conditions is due to heat storage within the lower body. These results may aid in understanding thermoregulatory control mechanisms of populations with a thermoregulatory dysfunction, such as those with spinal cord injuries.  相似文献   

20.
Abstract

The effect of active and passive recovery on repeated-sprint swimming bouts was studied in eight elite swimmers. Participants performed three trials of two sets of front crawl swims with 5 min rest between sets. Set A consisted of four 30-s bouts of high-intensity tethered swimming separated by 30 s passive rest, whereas Set B consisted of four 50-yard maximal-sprint swimming repetitions at intervals of 2 min. Recovery was active only between sets (AP trial), between sets and repetitions of Set B (AA trial) or passive throughout (PP trial). Performance during and metabolic responses after Set A were similar between trials. Blood lactate concentration after Set B was higher and blood pH was lower in the PP (18.29 ± 1.31 mmol · l?1 and 7.12 ± 0.11 respectively) and AP (17.56 ± 1.22 mmol · l?1 and 7.14 ± 0.11 respectively) trials compared with the AA (14.13 ± 1.56 mmol · l?1 and 7.23 ± 0.10 respectively) trial (P < 0.01). Performance time during Set B was not different between trials (P > 0.05), but the decline in performance during Set B of the AP trial was less marked than in the AA or PP trials (main effect of sprints, P < 0.05). Results suggest that active recovery (60% of the 100-m pace) could be beneficial between training sets, and may compromise swimming performance between repetitions when recovery durations are short (< 2 min).  相似文献   

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