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1.
The effects of sodium phosphate and caffeine supplementation were assessed on repeated-sprint ability. Using a randomised, double-blind, Latin-square design, 12 female, team-sport players participated in four trials: (1) sodium phosphate and caffeine, (2) sodium phosphate and placebo (for caffeine), (3) caffeine and placebo (for sodium phosphate) and (4) placebo (for sodium phosphate and caffeine), with ~21 days separating each trial. After each trial, participants performed a simulated team-game circuit (4 × 15 min quarters) with 6 × 20-m repeated-sprints performed once before (Set 1), at half-time (Set 2), and after end (Set 3). Total sprint times were faster after sodium phosphate and caffeine supplementation compared with placebo (Set 1: = 0.003; Set 2: = ?0.51; Set 3: < 0.001; overall: = 0.020), caffeine (Set 3: = 0.004; overall: = 0.033) and sodium phosphate (Set 3: = ?0.67). Furthermore, total sprint times were faster after sodium phosphate supplementation compared with placebo (Set 1: = ?0.52; Set 3: = ?0.58). Best sprint results were faster after sodium phosphate and caffeine supplementation compared with placebo (Set 3: = 0.007, = ?0.90) and caffeine (Set 3: = 0.024, = ?0.73). Best sprint times were also faster after sodium phosphate supplementation compared with placebo (= ?0.54 to ?0.61 for all sets). Sodium phosphate and combined sodium phosphate and caffeine loading improved repeated-sprint ability.  相似文献   

2.
This study compares test-retest reliability and peak exercise responses from ramp-incremented (RAMP) and maximal perceptually-regulated (PRETmax) exercise tests during arm crank exercise in individuals reliant on manual wheelchair propulsion (MWP). Ten untrained participants completed four trials over 2-weeks (two RAMP (0–40 W + 5–10 W · min?1) trials and two PRETmax. PRETmax consisted of five, 2-min stages performed at Ratings of Perceived Exertion (RPE) 11, 13, 15, 17 and 20). Participants freely changed the power output to match the required RPE. Gas exchange variables, heart rate, power output, RPE and affect were determined throughout trials. The V?O2peak from RAMP (14.8 ± 5.5 ml · kg?1 · min?1) and PRETmax (13.9 ± 5.2 ml · kg?1 · min?1) trials were not different (P = 0.08). Measurement error was 1.7 and 2.2 ml · kg?1 · min?1 and coefficient of variation 5.9% and 8.1% for measuring V?O2peak from RAMP and PRETmax, respectively. Affect was more positive at RPE 13 (P = 0.02), 15 (P = 0.01) and 17 (P = 0.01) during PRETmax. Findings suggest that PRETmax can be used to measure V?O2peak in participants reliant on MWP and leads to a more positive affective response compared to RAMP.  相似文献   

3.
Abstract

It is not known if ergogenic effects of caffeine ingestion in athletic groups occur in the sedentary. To investigate this, we used a counterbalanced, double-blind, crossover design to examine the effects of caffeine ingestion (6 mg · kg?1 body-mass) on exercise performance, substrate utilisation and perceived exertion during 30 minutes of self-paced stationary cycling in sedentary men. Participants performed two trials, one week apart, after ingestion of either caffeine or placebo one hour before exercise. Participants were instructed to cycle as quickly as they could during each trial. External work (J · kg?1) after caffeine ingestion was greater than after placebo (P = 0.001, effect size [ES] = 0.3). Further, heart rate, oxygen uptake and energy expenditure during exercise were greater after caffeine ingestion (P = 0.031, ES = 0.4; P = 0.009, ES = 0.3 and P = 0.018, ES = 0.3; respectively), whereas ratings of perceived exertion and respiratory exchange ratio values did not differ between trials (P = 0.877, ES = 0.1; P = 0.760, ES = 0.1; respectively). The ability to do more exercise after caffeine ingestion, without an accompanying increase in effort sensation, could motivate sedentary men to participate in exercise more often and so reduce adverse effects of inactivity on health.  相似文献   

4.
Abstract

This study investigated the effects of acute caffeine ingestion following short-term creatine supplementation on an incremental cycling to exhaustion task. Twelve active males performed the task under three conditions: baseline condition (BASE, no ergogenic aid), creatine plus caffeine condition (CRE + CAF), and creatine with placebo condition (CRE + PLA). Following the establishment of BASE condition, participants were administered CRE + CAF (0.3 g·kg?1·day?1 of creatine for 5 days followed by 6 mg·kg?1 of caffeine 1 h prior to testing) and CRE + PLA (0.3 g·kg?1·day?1 of creatine for 5 days followed by 6 mg·kg?1 of placebo 1 h prior to testing) in a double-blind, randomized crossover and counterbalancing protocol. No significant differences were observed in relative maximal oxygen consumption ([Vdot]O2max) (51.7±5.5, 52.8±4.9 and 51.3±5.6 ml·kg?1·min?1 for BASE, CRE + CAF and CRE + PLA, respectively; P>0.05) and absolute [Vdot]O2max (3.6±0.4, 3.7±0.4 and 3.5±0.5 l·min?1 for BASE, CRE + CAF and CRE + PLA, respectively; P>0.05). Blood samples indicated significantly higher blood lactate and glucose concentrations in the CRE + CAF among those in the BASE or CRE + PLA condition during the test (P<0.05). The time to exhaustion on a cycling ergometer was significantly longer for CRE + CAF (1087.2±123.9 s) compared with BASE (1009.2±86.0 s) or CRE + PLA (1040.3±96.1 s). This study indicated that a single dose of caffeine following short-term creatine supplementation did not hinder the creatine–caffeine interaction. In fact, it lengthened the time to exhaustion during an incremental maximum exercise test. However, this regime might lead to the accumulation of lactate in the blood.  相似文献   

5.
We examined the influence of caffeine supplementation on cognitive performance and perceptual responses in female team-game players taking low-dose monophasic oral contraceptives of the same hormonal composition. Ten females (24 ± 4 years; 59.7 ± 3.5 kg body mass; 2–6 training sessions per week) took part in a randomised, double-blind, placebo-controlled crossover-design trial. A 90-min intermittent treadmill-running protocol was completed 60 min following ingestion of a capsule containing either 6 mg ? kg?1 anhydrous caffeine or artificial sweetener (placebo). Perceptual responses (ratings of perceived exertion (RPE), feeling scale (FS), felt arousal scale (FAS)), mood (profile of mood states (POMS)) and cognitive performance (Stroop test, choice reaction time (CRT)) were completed before, during and after the exercise protocol, as well as after ~12 h post exercise. Caffeine ingestion significantly enhanced the ratings of pleasure (= 0.008) and arousal (= 0.002) during the exercise protocol, as well as increased vigour (POMS; = 0.007), while there was a tendency for reduced fatigue (POMS; = 0.068). Caffeine ingestion showed a tendency to decrease RPE (= 0.068) and improve reaction times in the Stroop (= 0.072) and CRT (= 0.087) tests. Caffeine supplementation showed a positive effect on perceptual parameters by increasing vigour and a tendency to decrease fatigue during intermittent running activity in female games players taking low-dose monophasic oral contraceptive steroids (OCS).  相似文献   

6.
Abstract

The aim of this study was to investigate the effects of caffeine supplementation on peak anaerobic power output (Wmax). Using a counterbalanced, randomised, double-blind, placebo-controlled design, 14 well-trained men completed three trials of a protocol consisting of a series of 6-s cycle ergometer sprints, separated by 5-min passive recovery periods. Sprints were performed at progressively increasing torque factors to determine the peak power/torque relationship and Wmax. Apart from Trial 1 (familiarisation), participants ingested a capsule containing 5 mg·kg?1 of caffeine or placebo, one hour before each trial. The effects of caffeine on blood lactate were investigated using capillary samples taken after each sprint. The torque factor which produced Wmax was not significantly different (p ≥ 0.05) between the caffeine (1.15 ± 0.08 N·m·kg?1) and placebo (1.13 ± 0.10 N·m·kg?1) trials. There was, however, a significant effect (p < 0.05) of supplementation on Wmax, with caffeine producing a higher value (1885 ± 303 W) than placebo (1835 ± 290 W). Analysis of the blood lactate data revealed a significant (p < 0.05) torque factor × supplement interaction with values being significantly higher from the sixth sprint (torque factor 1.0 N·m·kg?1) onwards following caffeine supplementation. The results of this study confirm previous reports that caffeine supplementation significantly increases blood lactate and Wmax. These findings may explain why the majority of previous studies, which have used fixed-torque factors of around 0.75 N·m·kg?1 and thereby failing to elicit Wmax, have failed to find an effect of caffeine on sprinting performance.  相似文献   

7.
8.
To assess the effect of carbohydrate and caffeine on gross efficiency (GE), 14 cyclists (V?O2max 57.6 ± 6.3 ml.kg?1.min?1) completed 4 × 2-hour tests at a submaximal exercise intensity (60% Maximal Minute Power). Using a randomized, counter-balanced crossover design, participants consumed a standardised diet in the 3-days preceding each test and subsequently ingested either caffeine (CAF), carbohydrate (CHO), caffeine+carbohydrate (CAF+CHO) or water (W) during exercise whilst GE and plasma glucose were assessed at regular intervals (~30 mins). GE progressively decreased in the W condition but, whilst caffeine had no effect, this was significantly attenuated in both trials that involved carbohydrate feedings (W = ?1.78 ± 0.31%; CHO = ?0.70 ± 0.25%, p = 0.008; CAF+CHO = ?0.63 ± 0.27%, p = 0.023; CAF = ?1.12 ± 0.24%, p = 0.077). Blood glucose levels were significantly higher in carbohydrate ingestion conditions (CHO = 4.79 ± 0.67 mmol·L?1, p < 0.001; CAF+CHO = 5.05 ± 0.81 mmol·L?1, p < 0.001; CAF = 4.46 ± 0.75 mmol·L?1; W = 4.20 ± 0.53 mmol·L?1). Carbohydrate ingestion has a small but significant effect on exercise-induced reductions in GE, indicating that cyclists’ feeding strategy should be carefully monitored prior to and during assessment.  相似文献   

9.
This investigation examined the effect of beetroot juice (BR) supplementation, a source of dietary nitrate (NO3?), on cycling time-trial (TT) performance and thermoregulation in the heat. In a double-blind, repeated-measures design, 12 male cyclists (age 26.6 ± 4.4 years, VO2peak 65.8 ± 5.5 mL.kg?1.min?1) completed four cycling TTs (14 kJ.kg?1) in hot (35°C, 48% relative humidity) and euthermic (21°C, 52%) conditions, following 3 days supplementation with BR (6.5 mmol NO3? for 2 days and 13 mmol NO3? on the final day), or NO3depleted placebo (PLA). Salivary NO3? and nitrite, core (Tc) and mean skin temperature (Tsk) were measured. Salivary NO3? and nitrite increased significantly post-BR supplementation (< 0.001). Average TT completion time (mm:ss) in hot conditions was 56:50 ± 05:08 with BR, compared with 58:30 ± 04:48 with PLA (= 0.178). In euthermic conditions, average completion time was 53:09 ± 04:35 with BR, compared with 54:01 ± 04:05 with PLA (= 0.380). The TT performance decreased (< 0.001), and Tc (< 0.001) and Tsk (< 0.001) were higher in hot compared with euthermic conditions. In summary, BR supplementation has no significant effect on cycling TT performance in the heat.  相似文献   

10.
This study investigated (i) whether the accumulated oxygen deficit (AOD) and curvature constant of the power–duration relationship (W′) are different during constant work-rate to exhaustion (CWR) and 3-min all-out (3MT) tests and (ii) the relationship between AOD and W′ during CWR and 3MT. Twenty-one male cyclists (age: 40 ± 6 years; maximal oxygen uptake [V?O2max]: 58 ± 7 ml · kg?1 · min?1) completed preliminary tests to determine the V?O2–power output relationship and V?O2max. Subsequently, AOD and W′ were determined as the difference between oxygen demand and oxygen uptake and work completed above critical power, respectively, in CWR and 3MT. There were no differences between tests for duration, work, or average power output (≥ 0.05). AOD was greater in the CWR test (4.18 ± 0.95 vs. 3.68 ± 0.98 L; = 0.004), whereas W′ was greater in 3MT (9.55 ± 4.00 vs. 11.37 ± 3.84 kJ; = 0.010). AOD and W′ were significantly correlated in both CWR (P < 0.001, r = 0.654) and 3MT (P < 0.001, r = 0.654). In conclusion, despite positive correlations between AOD and W′ in CWR and 3MT, between-test differences in the magnitude of AOD and W′, suggest that both measures have different underpinning mechanisms.  相似文献   

11.
Abstract

The aims of this study were two-fold: (1) to consider the criterion-related validity of the multi-stage fitness test (MSFT) by comparing the predicted maximal oxygen uptake ([Vdot]O2max) and distance travelled with peak oxygen uptake ([Vdot]O2peak) measured using a wheelchair ergometer (n = 24); and (2) to assess the reliability of the MSFT in a sub-sample of wheelchair athletes (n = 10) measured on two occasions. Twenty-four trained male wheelchair basketball players (mean age 29 years, s = 6) took part in the study. All participants performed a continuous incremental wheelchair ergometer test to volitional exhaustion to determine [Vdot]O2peak, and the MSFT on an indoor wooden basketball court. Mean ergometer [Vdot]O2peak was 2.66 litres · min?1 (s = 0.49) and peak heart rate was 188 beats · min?1 (s = 10). The group mean MSFT distance travelled was 2056 m (s = 272) and mean peak heart rate was 186 beats · min?1 (s = 11). Low to moderate correlations (ρ = 0.39 to 0.58; 95% confidence interval [CI]: ?0.02 to 0.69 and 0.23 to 0.80) were found between distance travelled in the MSFT and different expressions of wheelchair ergometer [Vdot]O2peak. There was a mean bias of ?1.9 beats · min?1 (95% CI: ?5.9 to 2.0) and standard error of measurement of 6.6 beats · min?1 (95% CI: 5.4 to 8.8) between the ergometer and MSFT peak heart rates. A similar comparison of ergometer and predicted MSFT [Vdot]O2peak values revealed a large mean systematic bias of 15.3 ml · kg?1 · min?1 (95% CI: 13.2 to 17.4) and standard error of measurement of 3.5 ml · kg?1 · min?1 (95% CI: 2.8 to 4.6). Small standard errors of measurement for MSFT distance travelled (86 m; 95% CI: 59 to 157) and MSFT peak heart rate (2.4 beats · min?1; 95% CI: 1.7 to 4.5) suggest that these variables can be measured reliably. The results suggest that the multi-stage fitness test provides reliable data with this population, but does not fully reflect the aerobic capacity of wheelchair athletes directly.  相似文献   

12.
Abstract

The aim of this study was to assess the effect of caffeine ingestion on 8 km run performance using an ecologically valid test protocol. A randomized double-blind crossover study was conducted involving eight male distance runners. The participants ran an 8 km race 1 h after ingesting a placebo capsule, a caffeine capsule (3 mg · kg?1 body mass) or no supplement. Heart rate was recorded at 5 s intervals throughout the race. Blood lactate concentration and ratings of perceived exertion were recorded after exercise. A repeated-measures analysis of variance (ANOVA) identified a significant treatment effect for 8 km performance time (P < 0.05); caffeine resulted in a mean improvement of 23.8 s (95% confidence interval [CI] = 13.1 to 34.5 s) in 8 km performance time (1.2% improvement, 95% CI = 0.7 to 1.8%). In addition, a two-way (time × condition) repeated-measures ANOVA identified a significantly higher blood lactate concentration 3 min after exercise during the caffeine trial (P < 0.05). We conclude that ingestion of 3 mg · kg?1 body mass of caffeine can improve absolute 8 km run performance in an ecologically valid race setting.  相似文献   

13.
This investigation assessed the effect of dietary nitrate (NO3?) supplementation, in the form of beetroot juice (BR), on repeat-sprint performance in normoxia and normobaric hypoxia. 12 male team-sport athletes (age 22.3 ± 2.6 y, VO2peak 53.1 ± 8.7 mL.kg?1.min?1) completed three exercise trials involving a 10 min submaximal warm-up and 4 sets of cycling repeat-sprint efforts (RSE; 9 × 4 s) at sea level (CON), or at 3000 m simulated altitude following acute supplementation (140 mL) with BR (HYPBR; 13 mmol NO3?) or NO3depleted BR placebo (HYPPLA). Peak (PPO) and mean (MPO) power output, plus work decrement were recorded during the RSE task, while oxygen consumption (VO2) was measured during the warm-up. There were no significant differences observed between HYPBR and HYPPLA for PPO or MPO; however, work decrement was reduced in the first RSE set in HYPBR compared with HYPPLA. There was a moderate effect for VO2 to be lower following BR at the end of the 10 min warm-up (ES = 0.50 ± 0.51). Dietary NO3? may not improve repeat-sprint performance in hypoxia but may reduce VO2 during submaximal exercise. Therefore, BR supplementation may be more effective for performance improvement during predominantly aerobic exercise.  相似文献   

14.
15.
Abstract

There is little published data in relation to the effects of caffeine upon cycling performance, speed and power in trained cyclists, especially during cycling of ~60 s duration. To address this, eight trained cyclists performed a 1 km time-trial on an electronically braked cycle ergometer under three conditions: after ingestion of 5 mg · kg?1 caffeine, after ingestion of a placebo, or a control condition. The three time-trials were performed in a randomized order and performance time, mean speed, mean power and peak power were determined. Caffeine ingestion resulted in improved performance time (caffeine vs. placebo vs. control: 71.1 ± 2.0 vs. 73.4 ± 2.3 vs. 73.3 ± 2.7 s; P = 0.02; mean ± s). This change represented a 3.1% (95% confidence interval: 0.7–5.6) improvement compared with the placebo condition. Mean speed was also higher in the caffeine than placebo and control conditions (caffeine vs. placebo vs. control: 50.7 ± 1.4 vs. 49.1 ± 1.5 vs. 49.2 ± 1.7 km · h?1; P = 0.0005). Mean power increased after caffeine ingestion (caffeine vs. placebo vs. control: 523 ± 43 vs. 505 ± 46 vs. 504 ± 38 W; P = 0.007). Peak power also increased from 864 ± 107 W (placebo) and 830 ± 87 W (control) to 940 ± 83 W after caffeine ingestion (P = 0.027). These results provide support for previous research that found improved performance after caffeine ingestion during short-duration high-intensity exercise. The magnitude of the improvements observed in our study could be due to our use of sport-specific ergometry, a tablet form and trained participants.  相似文献   

16.
Abstract

In this study, we examined fat oxidation rates during exercise in obese pubescent boys. Three groups of pubescent boys (16 pre-pubescent, Tanner's stage I; 16 pubescent, Tanner's stage III; and 14 post-pubescent, Tanner's stage V) performed a graded test on a leg cycle ergometer. The first step of the test was fixed at 30 W and power was gradually increased by 20 W every 3.5 min. Oxygen consumption ([Vdot]O2) and carbon dioxide production ([Vdot]CO2) were determined as the means of measurements during the last 30 s of each step, which allowed us to calculate fat oxidation rates versus exercise intensity. Between 20 and 50% of peak oxygen consumption ([Vdot]O2peak), fat oxidation rate in relative values (mg · min?1 · kg FFM?1) decreased continuously with pubertal development. In the same way, the maximum rate of fat oxidation occurred at a lower percentage of [Vdot]O2peak (pre-pubescent: 49.47 ± 1.62%; pubescent: 47.43 ± 1.26%; post-pubescent: 45.00 ± 0.97%). Our results confirm that puberty is responsible for a decrease in fat free mass capacities to use fat during exercise. The results suggest that post-pubescent obese boys need to practise physical activity at a lower intensity than pre-pubescent boys to enhance lipolysis and diminish adipose tissue and the consequences of obesity.  相似文献   

17.
Abstract

The single-stage treadmill walking test of Ebbeling et al. is commonly used to predict maximal oxygen consumption ([Vdot]O2max) from a submaximal effort between 50% and 70% of the participant's age-predicted maximum heart rate. The purpose of this study was to determine if this submaximal test correctly predicts [Vdot]O2max at the low (50% of maximum heart rate) and high (70% of maximum heart rate) ends of the specified heart rate range for males and females aged 18 – 55 years. Each of the 34 participants completed one low-intensity and one high-intensity trial. The two trials resulted in significantly different estimates of [Vdot]O2max (low-intensity trial: mean 40.5 ml · kg?1 · min?1, s = 9.3; high-intensity trial: 47.5 ml · kg?1 · min?1, s = 8.8; P < 0.01). A subset of 22 participants concluded their second trial with a [Vdot]O2max test (mean 47.9 ml · kg?1 · min?1, s = 8.9). The low-intensity trial underestimated (mean difference = ?3.5 ml · kg?1 · min?1; 95% CI = ?6.4 to ?0.6 ml · kg?1 · min?1; P = 0.02) and the high-intensity trial overestimated (mean difference = 3.5 ml · kg?1 · min?1; 95% CI = 1.1 to 6.0 ml · kg?1 · min?1; P = 0.01) the measured [Vdot]O2max. The predictive validity of Ebbeling and colleagues' single-stage submaximal treadmill walking test is diminished when performed at the extremes of the specified heart rate range.  相似文献   

18.
Abstract

The current study implemented a two-part design to (1) assess the vitamin D concentration of a large cohort of non-vitamin D supplemented UK-based athletes and 30 age-matched healthy non-athletes and (2) to examine the effects of 5000 IU · day?1 vitamin D3 supplementation for 8-weeks on musculoskeletal performance in a placebo controlled trial. Vitamin D concentration was determined as severely deficient if serum 25(OH)D < 12.5 nmol · l?1, deficient 12.5–30 nmol · l?1 and inadequate 30–50 nmol · l?1. We demonstrate that 62% of the athletes (38/61) and 73% of the controls (22/30) exhibited serum total 25(OH)D < 50 nmol · l?1. Additionally, vitamin D supplementation increased serum total 25(OH)D from baseline (mean ± SD = 29 ± 25 to 103 ± 25 nmol · l?1, P = 0.0028), whereas the placebo showed no significant change (53 ± 29 to 74 ± 24 nmol · l?1, P = 0.12). There was a significant increase in 10 m sprint times (P = 0.008) and vertical-jump (P = 0.008) in the vitamin D group whereas the placebo showed no change (P = 0.587 and P = 0.204 respectively). The current data supports previous findings that athletes living at Northerly latitudes (UK = 53° N) exhibit inadequate vitamin D concentrations (<50 nmol · l?1). Additionally the data suggests that inadequate vitamin D concentration is detrimental to musculoskeletal performance in athletes. Future studies using larger athletic groups are now warranted.  相似文献   

19.
Abstract

Ten healthy, non-cycling trained males (age: 21.2 ± 2.2 years, body mass: 75.9 ± 13.4 kg, height: 178 ± 6 cm, [Vdot]O2PEAK: 46 ± 10 ml · kg?1 · min?1) performed a graded incremental exercise test, two familiarisation trials and six experimental trials. Experimental trials consisted of cycling to volitional exhaustion at 100%, 110% and 120% WPEAK, 60 min after ingesting either 0.3 g · kg?1 body mass sodium bicarbonate (NaHCO3) or 0.1 g · kg?1 body mass sodium chloride (placebo). NaHCO3 ingestion increased cycling capacity by 17% at 100% WPEAK (327 vs. 383 s; P = 0.02) although not at 110% WPEAK (249 vs. 254 s; P = 0.66) or 120% WPEAK (170 vs. 175 s; P = 0.60; placebo and NaHCO3 respectively). Heart rate (P = 0.02), blood lactate (P = 0.001), pH (P < 0.001), [HCO3 ?], (P < 0.001), and base excess (P < 0.001) were greater in all NaHCO3 trials. NaHCO3 attenuated localised ratings of perceived exertion (RPEL) to a greater extent than placebo only at 100% WPEAK (P < 0.02). Ratings of abdominal discomfort and gut fullness were mild but higher for NaHCO3. NaHCO3 ingestion significantly improves continuous constant load cycling at 100% WPEAK due to, in part, attenuation of RPEL.  相似文献   

20.
Plasma heat shock protein 70 (HSP70) concentrations rise during heat stress, which can independently induce cytokine production. Upper body exercise normally results in modest body temperature elevations. The aim of this study was to investigate the impacts of additional clothing on the body temperature, cytokine and HSP70 responses during this exercise modality. Thirteen males performed 45-min constant-load arm cranking at 63% maximum aerobic power (62 ± 7%V?O2peak) in either a non-permeable whole-body suit (intervention, INT) or shorts and T-shirt (control, CON). Exercise resulted in a significant increase of IL-6 and IL-1ra plasma concentrations (< 0.001), with no difference between conditions (> 0.19). The increase in HSP70 from pre to post was only significant for INT (0.12 ± 0.11ng?mL?1, < 0.01 vs. 0.04 ± 0.18 ng?mL?1, = 0.77). Immediately following exercise, Tcore was elevated by 0.46 ± 0.29 (INT) and 0.37 ± 0.23ºC (CON), respectively (< 0.01), with no difference between conditions (= 0.16). The rise in mean Tskin (2.88 ± 0.50 and 0.30 ± 0.89ºC, respectively) and maximum heat storage (3.24 ± 1.08 and 1.20 ± 1.04 J?g?1, respectively) was higher during INT (< 0.01). Despite large differences in heat storage between conditions, the HSP70 elevations during INT, even though significant, were very modest. Possibly, the Tcore elevations were too low to induce a more pronounced HSP70 response to ultimately affect cytokine production.  相似文献   

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