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1.
The aim of this study was to assess the responses of blood lactate and pyruvate during the lactate minimum speed test. Ten participants (5 males, 5 females; mean +/- s: age 27.1 +/- 6.7 years, VO 2max 52.0 +/- 7.9 ml kg -1 min -1 ) completed: (1) the lactate minimum speed test, which involved supramaximal sprint exercise to invoke a metabolic acidosis before the completion of an incremental treadmill test (this results in a ‘U-shaped’ blood lactate profile with the lactate minimum speed being defined as the minimum point on the curve); (2) a standard incremental exercise test without prior sprint exercise for determination of the lactate threshold; and (3) the sprint exercise followed by a passive recovery. The lactate minimum speed (12.0 +/- 1.4 km h -1 ) was significantly slower than running speed at the lactate threshold (12.4 +/- 1.7 km h -1 ) (P < 0.05), but there were no significant differences in VO 2 , heart rate or blood lactate concentration between the lactate minimum speed and running speed at the lactate threshold. During the standard incremental test, blood lactate and the lactate-topyruvate ratio increased above baseline values at the same time, with pyruvate increasing above baseline at a higher running speed. The rate of lactate, but not pyruvate, disappearance was increased during exercising recovery (early stages of the lactate minimum speed incremental test) compared with passive recovery. This caused the lactate-to-pyruvate ratio to fall during the early stages of the lactate minimum speed test, to reach a minimum point at a running speed that coincided with the lactate minimum speed and that was similar to the point at which the lactate-to-pyruvate ratio increased above baseline in the standard incremental test. Although these results suggest that the mechanism for blood lactate accumulation at the lactate minimum speed and the lactate threshold may be the same, disruption to normal submaximal exercise metabolism as a result of the preceding sprint exercise, including a three- to five-fold elevation of plasma pyruvate concentration, makes it difficult to interpret the blood lactate response to the lactate minimum speed test. Caution should be exercised in the use of this test for the assessment of endurance capacity.  相似文献   

2.
The aim of this study was to assess the sensitivity of the lactate minimum speed test to changes in endurance fitness resulting from a 6 week training intervention. Sixteen participants (mean +/- s: age 23+/-4 years; body mass 69.7+/-9.1 kg) completed 6 weeks of endurance training. Another eight participants (age 23+/-4 years; body mass 72.7+/-12.5 kg) acted as non-training controls. Before and after the training intervention, all participants completed: (1) a standard multi-stage treadmill test for the assessment of VO2max, running speed at the lactate threshold and running speed at a reference blood lactate concentration of 3 mmol x l(-1); and (2) the lactate minimum speed test, which involved two supramaximal exercise bouts and an 8 min walking recovery period to increase blood lactate concentration before the completion of an incremental treadmill test. Additionally, a subgroup of eight participants from the training intervention completed a series of constant-speed runs for determination of running speed at the maximal lactate steady state. The test protocols were identical before and after the 6 week intervention. The control group showed no significant changes in VO2max, running speed at the lactate threshold, running speed at a blood lactate concentration of 3 mmol x l(-1) or the lactate minimum speed. In the training group, there was a significant increase in VO2max (from 47.9+/-8.4 to 52.2+/-2.7 ml x kg(-1) x min(-1)), running speed at the maximal lactate steady state (from 13.3+/-1.7 to 13.9+/-1.6 km x h(-1)), running speed at the lactate threshold (from 11.2+/-1.8 to 11.9+/-1.8 km x h(-1)) and running speed at a blood lactate concentration of 3 mmol x l(-1) (from 12.5+/-2.2 to 13.2+/-2.1 km x h(-1)) (all P < 0.05). Despite these clear improvements in aerobic fitness, there was no significant difference in lactate minimum speed after the training intervention (from 11.0+/-0.7 to 10.9+/-1.7 km x h(-1)). The results demonstrate that the lactate minimum speed, when assessed using the same exercise protocol before and after 6 weeks of aerobic exercise training, is not sensitive to changes in endurance capacity.  相似文献   

3.
The aim of this study was to assess the sensitivity of the lactate minimum speed test to changes in endurance fitness resulting from a 6 week training intervention. Sixteen participants (mean +/- s :age 23 +/- 4 years;body mass 69.7 +/- 9.1 kg) completed 6 weeks of endurance training. Another eight participants (age 23 +/- 4 years; body mass 72.7 +/-12.5 kg) acted as non-training controls. Before and after the training intervention, all participants completed: (1) a standard multi-stage treadmill test for the assessment of VO 2max , running speed at the lactate threshold and running speed at a reference blood lactate concentration of 3 mmol.l -1 ; and (2) the lactate minimum speed test, which involved two supramaximal exercise bouts and an 8 min walking recovery period to increase blood lactate concentration before the completion of an incremental treadmill test. Additionally, a subgroup of eight participants from the training intervention completed a series of constant-speed runs for determination of running speed at the maximal lactate steady state. The test protocols were identical before and after the 6 week intervention. The control group showed no significant changes in VO 2max , running speed at the lactate threshold, running speed at a blood lactate concentration of 3 mmol.l -1 or the lactate minimum speed.In the training group, there was a significant increase in VO 2max (from 47.9 +/- 8.4 to 52.2 +/- 2.7 ml.kg -1 .min -1 ), running speed at the maximal lactate steady state (from 13.3 +/- 1.7 to 13.9 +/- 1.6 km.h -1 ), running speed at the lactate threshold (from 11.2 +/- 1.8 to 11.9 +/- 1.8 km.h -1 ) and running speed at a blood lactate concentration of 3 mmol.l -1 (from 12.5 +/- 2.2 to 13.2 +/- 2.1 km.h -1 ) (all P ? 0.05). Despite these clear improvements in aerobic fitness, there was no significant difference in lactate minimum speed after the training intervention (from 11.0 +/- 0.7 to 10.9 +/- 1.7 km.h -1 ). The results demonstrate that the lactate minimum speed,when assessed using the same exercise protocol before and after 6 weeks of aerobic exercise training, is not sensitive to changes in endurance capacity.  相似文献   

4.
It is common for the physiological working capacity of a triathlete when cycling and running to be assessed on two separate days. The aim of this study was to establish whether an incremental running test to exhaustion has a negative effect after a 5 h recovery from an incremental cycling test. Eight moderately trained triathletes (age, 26.2 +/- 3.4 years; body mass, 67.3 +/- 9.1 kg; VO2max when cycling, 59 +/- 13 ml x kg x min(-1); mean +/- s) completed an incremental running test 5 h after an incremental cycling test (fatigue) as well as an incremental running test without previous activity (control). Maximum running speed, maximal oxygen uptake (VO2max) and the lactate threshold were determined for each incremental running test and correlated with the average speed during a 5 km run, which was performed immediately after a 20 km cycling time-trial, as in a sprint triathlon. There were no significant differences in maximum running speed, VO2max or the lactate threshold in either incremental running test (control or fatigue). Furthermore, good agreement was found for each physiological variable in both the control and fatigue tests. For the fatigue test, there were significant correlations between the average speed during a 5 km run and both VO2max expressed in absolute terms (r = 0.83) and the lactate threshold (r = 0.88). However, maximum running speed correlated most strongly with the average speed during a 5 km run (r = 0.96). The results of this study indicate that, under controlled conditions, an incremental running test can be performed successfully 5 h after an incremental cycling test to exhaustion. Also, the maximum running speed achieved during an incremental running test is the variable that correlates most strongly with the average running speed during a 5 km run after a 20 km cycling time-trial in well-trained triathletes.  相似文献   

5.
The aims of this study were: (1) to identify the exercise intensity that corresponds to the maximal lactate steady state in adolescent endurance-trained runners; (2) to identify any differences between the sexes; and (3) to compare the maximal lactate steady state with commonly cited fixed blood lactate reference parameters. Sixteen boys and nine girls volunteered to participate in the study. They were first tested using a stepwise incremental treadmill protocol to establish the blood lactate profile and peak oxygen uptake (VO2). Running speeds corresponding to fixed whole blood lactate concentrations of 2.0, 2.5 and 4.0 mmol x l(-1) were calculated using linear interpolation. The maximal lactate steady state was determined from four separate 20-min constant-speed treadmill runs. The maximal lactate steady state was defined as the fastest running speed, to the nearest 0.5 km x h(-1), where the change in blood lactate concentration between 10 and 20 min was < 0.5 mmol x l(-1). Although the boys had to run faster than the girls to elicit the maximal lactate steady state (15.7 vs 14.3 km x h(-1), P < 0.01), once the data were expressed relative to percent peak VO2 (85 and 85%, respectively) and percent peak heart rate (92 and 94%, respectively), there were no differences between the sexes (P > 0.05). The running speed and percent peak VO2 at the maximal lactate steady state were not different to those corresponding to the fixed blood lactate concentrations of 2.0 and 2.5 mmol x l(-1) (P > 0.05), but were both lower than those at the 4.0 mmol x l(-1) concentration (P < 0.05). In conclusion, the maximal lactate steady state corresponded to a similar relative exercise intensity as that reported in adult athletes. The running speed, percent peak VO2 and percent peak heart rate at the maximal lactate steady state are approximated by the fixed blood lactate concentration of 2.5 mmol x l(-1) measured during an incremental treadmill test in boys and girls.  相似文献   

6.
We tested the hypothesis that exercise-induced muscle damage would increase the ventilatory (V(E)) response to incremental/ramp cycle exercise (lower the gas exchange threshold) without altering the blood lactate profile, thereby dissociating the gas exchange and lactate thresholds. Ten physically active men completed maximal incremental cycle tests before (pre) and 48 h after (post) performing eccentric exercise comprising 100 squats. Pulmonary gas exchange was measured breath-by-breath and fingertip blood sampled at 1-min intervals for determination of blood lactate concentration. The gas exchange threshold occurred at a lower work rate (pre: 136 ± 27 W; post: 105 ± 19 W; P < 0.05) and oxygen uptake (VO(2)) (pre: 1.58 ± 0.26 litres · min(-1); post: 1.41 ± 0.14 litres · min(-1); P < 0.05) after eccentric exercise. However, the lactate threshold occurred at a similar work rate (pre: 161 ± 19 W; post: 158 ± 22 W; P > 0.05) and VO(2) (pre: 1.90 ± 0.20 litres · min(-1); post: 1.88 ± 0.15 litres · min(-1); P > 0.05) after eccentric exercise. These findings demonstrate that exercise-induced muscle damage dissociates the V(E) response to incremental/ramp exercise from the blood lactate response, indicating that V(E) may be controlled by additional or altered neurogenic stimuli following eccentric exercise. Thus, due consideration of prior eccentric exercise should be made when using the gas exchange threshold to provide a non-invasive estimation of the lactate threshold.  相似文献   

7.
The aims of this study were to describe and determine the test-retest reliability of an exercise protocol, the Loughborough Intermittent Shuttle Test (the LIST), which was designed to simulate the activity pattern characteristic of the game of soccer. The protocol consisted of two parts: Part A comprised a fixed period of variable-intensity shuttle running over 20 m; Part B consisted of continuous running, alternating every 20 m between 55% and 95% VO2max, until volitional fatigue. Seven trained games players (age 21.5+/-0.9 years, height 182+/-2 cm, body mass 80.1+/-3.6 kg, VO2max 59.0+/-1.9 ml x kg(-1) x min(-1); mean +/- s(x)) performed the test on two occasions (Trial 1 and Trial 2), at least 7 days apart, to determine the test-retest reliability of the sprint times and running capacity. The physiological and metabolic responses on both occasions were also monitored. The participants ingested water ad libitum during the first trial, and were then prescribed the same amount of water during the second trial. The 15 m sprint times during Trials 1 and 2 averaged 2.42+/-0.04 s and 2.43+/-0.04 s, respectively. Run time during Part B was 6.3+/-2.0 min for Trial 1 and 6.1+/-1.3 min for Trial 2. The 95% limits of agreement for sprint times and run times during Part B were -0.14 to 0.12 s and -3.19 to 2.16 min respectively. There were no differences between trials for heart rate, rating of perceived exertion, body mass change during exercise, or blood lactate and glucose concentrations during the test. Thus, we conclude that the sprint times and the Part B run times were reproducible within the limits previously stated. In addition, the activity pattern and the physiological and metabolic responses closely simulated the match demands of soccer.  相似文献   

8.
The aim of this study was to determine the incidence of subject drop-out on a multi-stage shuttle run test and a modified incremental shuttle run test in which speed was increased by 0.014 m x s(-1) every 20-m shuttle to avoid the need for verbal speed cues. Analysis of the multi-stage shuttle run test with 208 elite female netball players and 381 elite male lacrosse players found that 13 (+/-3) players stopped after the first shuttle of each new level, in comparison with 5 (+/-2) players on any other shuttle. No obvious drop-out pattern was observed on the incremental shuttle run test with 273 male and 79 female undergraduate students. The mean difference between a test-retest condition (n = 20) for peak shuttle running speed (-0.03+/-0.01 m x s(-1)) and maximal heart rate (0.4+/-0.1 beats x min(-1)) on the incremental test showed no bias (P > 0.05). The 95% absolute confidence limits of agreement were+/-0.11 m x s(-1) for peak shuttle running speed and+/-5 beats min(-1) for maximal heart rate. The relationship (n = 27) between peak shuttle running speed on the incremental shuttle run test (4.22+/-0.14 m x s(-1)) and VO2max (59.0+/-1.7 ml kg(-1) x min(-1)) was r= 0.91 (P< 0.01), with a standard error of prediction of +/-2.6 ml x kg(-1) x min(-1). These results suggest verbal cues during the multi-stage shuttle run test may influence subject drop-out. The incremental shuttle run test shows no obvious drop-out patten and provides a valid estimate of VO2max.  相似文献   

9.
Comparison of maximal anaerobic running tests on a treadmill and track   总被引:1,自引:0,他引:1  
To develop a track version of the maximal anaerobic running test, 10 sprint runners and 12 distance runners performed the test on a treadmill and on a track. The treadmill test consisted of incremental 20-s runs with a 100-s recovery between the runs. On the track, 20-s runs were replaced by 150-m runs. To determine the blood lactate versus running velocity curve, fingertip blood samples were taken for analysis of blood lactate concentration at rest and after each run. For both the treadmill and track protocols, maximal running velocity (v max), the velocities associated with blood lactate concentrations of 10 mmol x l-1 (v10 mM) and 5 mmol x l(-1) (v5 mM), and the peak blood lactate concentration were determined. The results of both protocols were compared with the seasonal best 400-m runs for the sprint runners and seasonal best 1000-m time-trials for the distance runners. Maximal running velocity was significantly higher on the track (7.57 +/- 0.79 m x s(-1)) than on the treadmill (7.13 +/- 0.75 m x s(-1)), and sprint runners had significantly higher vmax, v10 mM, and peak blood lactate concentration than distance runners (P < 0.05). The Pearson product--moment correlation coefficients between the variables for the track and treadmill protocols were 0.96 (v max), 0.82 (v10 mM), 0.70 (v5 mM), and 0.78 (peak blood lactate concentration) (P < 0.05). In sprint runners, the velocity of the seasonal best 400-m run correlated positively with vmax in the treadmill (r = 0.90, P < 0.001) and track protocols (r = 0.92, P < 0.001). In distance runners, a positive correlation was observed between the velocity of the 1000-m time-trial and vmax in the treadmill (r = 0.70, P < 0.01) and track protocols (r = 0.63, P < 0.05). It is apparent that the results from the track protocol are related to, and in agreement with, the results of the treadmill protocol. In conclusion, the track version of the maximal anaerobic running test is a valid means of measuring different determinants of sprint running performance.  相似文献   

10.
The purpose of this study was to provide a more detailed analysis of performance in cross-country skiing by combining findings from a differential global positioning system (dGPS), metabolic gas measurements, speed in different sections of a ski-course and treadmill threshold data. Ten male skiers participated in a freestyle skiing field test (5.6 km), which was performed with dGPS and metabolic gas measurements. A treadmill running threshold test was also performed and the following parameters were derived: anaerobic threshold, threshold of decompensated metabolic acidosis, respiratory exchange ratio = 1, onset of blood lactate accumulation and peak oxygen uptake (VO2peak). The combined dGPS and metabolic gas measurements made detailed analysis of performance possible. The strongest correlations between the treadmill data and final skiing field test time were for VO2peak (l x min(-1)), respiratory exchange ratio = 1 (l x min(-1)) and onset of blood lactate accumulation (l x min(-1)) (r = -0.644 to - 0.750). However, all treadmill test data displayed stronger associations with speed in different stretches of the course than with final time, which stresses the value of a detailed analysis of performance in cross-country skiing. Mean oxygen uptake (VO2) in a particular stretch in relation to speed in the same stretch displayed its strongest correlation coefficients in most stretches when VO2 was presented in units litres per minute, rather than when VO2 was normalized to body mass (ml x kg(-1) x min(-1) and ml x min(-1) x kg(-2/3)). This suggests that heavy cross-country skiers have an advantage over their lighter counterparts. In one steep uphill stretch, however, VO2 (ml x min(-1) x kg(-2/3)) displayed the strongest association with speed, suggesting that in steep uphill sections light skiers could have an advantage over heavier skiers.  相似文献   

11.
The aim of this study was to predict indoor rowing performance in 12 competitive female rowers (age 21.3 +/- 3.6 years, height 1.68 +/- 0.54 m, body mass 67.1 +/- 11.7 kg; mean +/- s) using a 30 s rowing sprint, maximal oxygen uptake and the blood lactate response to submaximal rowing. Blood lactate and oxygen uptake (VO2) were measured during a discontinuous graded exercise test on a Concept II rowing ergometer incremented by 25 W for each 2 min stage; the highest VO2 measured during the test was recorded as VO2max (mean = 3.18 +/- 0.35 l.min-1). Peak power (380 +/- 63.2 W) and mean power (368 +/- 60.0 W) were determined using a modified Wingate test protocol on the Concept II rowing ergometer. Rowing performance was based on the results of the 2000 m indoor rowing championship in 1997 (466.8 +/- 12.3 s). Laboratory testing was performed within 3 weeks of the rowing championship. Submitting mean power (Power), the highest and lowest five consecutive sprint power outputs (Maximal and Minimal), percent fatigue in the sprint test (Fatigue), VO2max (l.min-1), VO2max (ml.kg-1.min-1), VO2 at the lactate threshold, power at the lactate threshold (W), maximal lactate concentration, lactate threshold (percent VO2max) and VEmax (l.min-1) to a stepwise multiple regression analysis produced the following model to predict 2000 m rowing performance: Time2000 = -0.163 (Power) -14.213.(VO2max l.min-1) +0.738.(Fatigue) 7.259 (R2 = 0.96, standard error = 2.89). These results indicate that, in the women studied, 75.7% of the variation in 2000 m indoor rowing performance time was predicted by peak power in a rowing Wingate test, while VO2max and fatigue during the Wingate test explained an additional 12.1% and 8.2% of the variance, respectively.  相似文献   

12.
Whole-body energy expenditure for heavy/severe exercise is currently accounted for by either: (1) anaerobic and oxygen uptake measures during exercise where recovery energy expenditure is omitted; or (2) oxygen uptake during, and an EPOC (excess post-exercise oxygen consumption), measure following exercise where substrate level phosphorylation during exercise is considered part of EPOC. Simultaneous direct/indirect calorimetry enabled us to determine if a thermodynamic reversal (i.e. heat consumption) takes place as the highly exothermic pyruvate to lactate reaction proceeds in the opposite direction. Reversibility implies that oxygen uptake (e.g. EPOC) can indeed account for rapid glycolytic ATP production regardless if lactate is formed or not (e.g. 1.2 g glucose catabolism = 20.9 kJ x l O2(-1)). Cultured hybrid cells and mouse cardiac muscle fibres were utilized in simultaneous calorimetry and respirometry experiments where pyruvate or lactate was predominantly oxidized. The calorimetric to respiratory ratio was determined using heat flux (pW x cell(-1)) and oxygen flux (pmol x s(-1) cell(-1)) measures. Ten cell experiments gave calorimetric to respiratory ratios that showed no statistical difference (P= 0.97) whether cells respired predominantly on lactate (-516+/-53 kJ x mol O2(-1)) or pyruvate (- 517+/-89 kJ x mol O2(-1)). In three cardiac preparations, the calorimetric to respiratory ratio was -502+/-15 kJ x mol O2(-1) for lactate and -506+/-47 kJ x mol O2(-1) for pyruvate, again a non-significant difference (P= 0.91). Heat consumption did not occur during lactate oxidation. These results suggest that rapid glycolytic ATP and lactate production, and lactate oxidation, are both independently associated with heat production and thus represent separate and additive components to the measurement of total energy expenditure for exercise and recovery.  相似文献   

13.
The aim of this study was to compare the cycling performance of cyclists and triathletes. Each week for 3 weeks, and on different days, 25 highly trained male cyclists and 18 highly trained male triathletes performed: (1) an incremental exercise test on a cycle ergometer for the determination of peak oxygen consumption (VO2peak), peak power output and the first and second ventilatory thresholds, followed 15 min later by a sprint to volitional fatigue at 150% of peak power output; (2) a cycle to exhaustion test at the VO2peak power output; and (3) a 40-km cycle time-trial. There were no differences in VO2peak, peak power output, time to volitional fatigue at 150% of peak power output or time to exhaustion at VO2peak power output between the two groups. However, the cyclists had a significantly faster time to complete the 40-km time-trial (56:18 +/- 2:31 min:s; mean +/- s) than the triathletes (58:57 +/- 3:06 min:s; P < 0.01), which could be partially explained (r = 0.34-0.51; P < 0.05) by a significantly higher first (3.32 +/- 0.36 vs 3.08 +/- 0.36 l x min(-1)) and second ventilatory threshold (4.05 +/- 0.36 vs 3.81 +/- 0.29 l x min(-1); both P < 0.05) in the cyclists compared with the triathletes. In conclusion, cyclists may be able to perform better than triathletes in cycling time-trial events because they have higher first and second ventilatory thresholds.  相似文献   

14.
The aims of this study were to describe and determine the test-retest reliability of an exercise protocol, the Loughborough Intermittent Shuttle Test (the LIST), which was designed to simulate the activity pattern characteristic of the game of soccer. The protocol consisted of two parts: Part A comprised a fixed period of variable-intensity shuttle running over 20 m; Part B consisted of continuous running, alternating every 20 m between 55% and 95% VO 2max , until volitional fatigue. Seven trained games players (age 21.5 +/- 0.9 years, height 182 +/- 2 cm, body mass 80.1 +/- 3.6 kg, VO 2max 59.0 +/- 1.9 ml kg -1 min -1 ; mean s x ) performed the test on two occasions (Trial 1 and Trial 2), at least7 days apart, to determine the test-retest reliability of the sprint times and running capacity. The physiological and metabolic responses on both occasions were also monitored. The participants ingested water ad libitum during the first trial, and were then prescribed the same amount of water during the second trial. The 15 m sprint times during Trials 1 and 2 averaged 2.42 +/- 0.04 s and 2.43 +/- 0.04 s, respectively. Run time during Part B was 6.3 +/- 2.0 min for Trial 1 and 6.1 +/- 1.3 min for Trial 2. The 95% limits of agreement for sprint times and run times during Part B were -0.14 to 0.12 s and -3.19 to 2.16 min respectively. There were no differences between trials for heart rate, rating of perceived exertion, body mass change during exercise, or blood lactate and glucose concentrations during the test. Thus, we conclude that the sprint times and the Part B run times were reproducible within the limits previously stated. In addition, the activity pattern and the physiological and metabolic responses closely simulated the match demands of soccer.  相似文献   

15.
16.
The aims of the study were to modify the training impulse (TRIMP) method of quantifying training load for use with intermittent team sports, and to examine the relationship between this modified TRIMP (TRIMP(MOD)) and changes in the physiological profile of team sport players during a competitive season. Eight male field hockey players, participating in the English Premier Division, took part in the study (mean+/-s: age 26+/-4 years, body mass 80.8+/-5.2 kg, stature 1.82+/-0.04 m). Participants performed three treadmill exercise tests at the start of the competitive season and mid-season: a submaximal test to establish the treadmill speed at a blood lactate concentration of 4 mmol . l(-1); a maximal incremental test to determine maximal oxygen uptake ([V]O(2max)) and peak running speed; and an all-out constant-load test to determine time to exhaustion. Heart rate was recorded during all training sessions and match-play, from which TRIMP(MOD) was calculated. Mean weekly TRIMP(MOD) was correlated with the change in [V]O(2max) and treadmill speed at a blood lactate concentration of 4 mmol x l(-1) from the start of to mid-season (P<0.05). The results suggest that TRIMP(MOD) is a means of quantifying training load in team sports and can be used to prescribe training for the maintenance or improvement of aerobic fitness during the competitive season.  相似文献   

17.
The aim of this study was to assess the effect of time of day on physiological responses to running at the speed at the lactate threshold. After determination of the lactate threshold, using a standard incremental protocol, nine male runners (age 26.3 +/- 5.7 years, height 1.77 +/- 0.07 m, mass 73.1 +/- 6.5 kg, lactate threshold speed 13.6 +/- 1.6 km x h(-1); mean +/- s) completed a standardized 30 min run at lactate threshold speed, twice within 24 h (07:00-09:00 h and 18:00-21:00 h). Core body temperature, heart rate, minute ventilation, oxygen uptake, carbon dioxide expired, respiratory exchange ratio and capillary blood lactate were measured at rest, after a warm-up and at 10, 20 and 30 min during the run. In addition, the rating of perceived exertion was reported every 10 min during the run. Significant diurnal variation was observed only for body temperature (36.9 +/- 0.9 degrees C vs 37.3 +/- 0.3 degrees C) and respiratory exchange ratio at rest (0.86 +/- 0.01 vs 0.89 +/- 0.07) (P < 0.05). Diurnal variation persisted for body temperature throughout the warm-up (37.1 +/- 0.2 degrees C vs 37.5 +/- 0.3 degrees C) and during exercise (36.2 +/- 0.6 degrees C vs 38.6 +/- 0.4 degrees C), but only during the warm-up for the respiratory exchange ratio (0.85 +/- 0.05 vs 0.87 +/- 0.02) (P < 0.05). The rating of perceived exertion was significantly elevated during the morning trial (12.7 +/- 0.9 vs 11.9 +/- 1.2) (P < 0.05). These findings suggest that, despite the diurnal variation in body temperature, other physiological responses to running at lactate threshold speed are largely unaffected. However, a longer warm-up may be required in morning trials because of a slower increase in body temperature, which could have an impact on ventilation responses and ratings of perceived exertion.  相似文献   

18.
It has previously been shown that measurement of the critical speed is a non-invasive method of estimating the blood lactate response during exercise. However, its validity in children has yet to be demonstrated. The aims of this study were: (1) to verify if the critical speed determined in accordance with the protocol of Wakayoshi et al. is a non-invasive means of estimating the swimming speed equivalent to a blood lactate concentration of 4 mmol x l(-1) in children aged 10-12 years; and (2) to establish whether standard of performance has an effect on its determination. Sixteen swimmers were divided into two groups: beginners and trained. They initially completed a protocol for determination of speed equivalent to a blood lactate concentration of 4 mmol x l(-1). Later, during training sessions, maximum efforts were swum over distances of 50, 100 and 200 m for the calculation of the critical speed. The speeds equivalent to a blood lactate concentration of 4 mmol x l(-1) (beginners = 0.82 +/- 0.09 m x s(-1), trained = 1.19 +/- 0.11 m x s(-1); mean +/- s) were significantly faster than the critical speeds (beginners = 0.78 +/- 0.25 m x s(-1), trained = 1.08 +/- 0.04 m x s(-1)) in both groups. There was a high correlation between speed at a blood lactate concentration of 4 mmol x l(-1) and the critical speed for the beginners (r= 0.96, P < 0.001), but not for the trained group (r= 0.60, P> 0.05). The blood lactate concentration corresponding to the critical speed was 2.7 +/- 1.1 and 3.1 +/- 0.4 mmol x l(-1) for the beginners and trained group respectively. The percent difference between speed at a blood lactate concentration of 4 mmol x l(-1) and the critical speed was not significantly different between the two groups. At all distances studied, swimming performance was significantly faster in the trained group. Our results suggest that the critical speed underestimates swimming intensity corresponding to a blood lactate concentration of 4 mmol x l(-1) in children aged 10-12 years and that standard of performance does not affect the determination of the critical speed.  相似文献   

19.
The aim of this study was to establish the validity of the breakpoint in breathing frequency for the assessment of the lactate threshold and the ventilatory threshold during incremental running exercise. Twelve trained runners (mean +/- s: age 29 +/- 8 years; body mass 68.7 8.8 kg; V O 2m ax 57.9 +/- 4.1 ml . kg -1 . min -1 ) performed randomly assigned incremental treadmill tests on separate days. In addition to the assessment of the V O 2m ax (Test 1), the subjects performed two standard multi-stage treadmill tests (4-min stages) for the assessment of the lactate threshold while wearing (Test 2) and not wearing (Test 3) a standard mouthpiece and noseclip arrangement. Breathing frequency was measured by a thermistor, which was positioned in the back of the mouthpiece for Test 2, and fixed 3 cm in front of the mouth using a headband and flexible wiring for Test 3. All exercise tests were recorded on videotape and mean breathing frequency and stride rate were calculated for the last minute of each stage from real-time playback of the videotapes. The breathing frequency breakpoint was determined in six subjects only for Test 2 and in five subjects only for Test 3. For Test 2, there were no differences between the breakpoint in breathing frequency (14.7 +/- 0.9 km . h - 1), the lactate threshold (14.7 +/- 0.9 km . h -1 ) and the ventilatory threshold (14.7 +/- 1.1 km . h -1 ). For Test 3, the breakpoint in breathing frequency (14.0 +/- 1.0 km . h -1 ) was not appreciably different from the lactate threshold (14.7 +/- 1.2 km . h -1 ). Hey plots showed marked interindividual differences in the responses of breathing frequency and tidal volume to exercise. In four subjects, the ventilatory threshold was mediated by a non-linear increase in tidal volume, with breathing frequency either increasing in a linear manner (n = 1) or remaining constant owing to entrainment of breathing frequency to cadence (n = 3). We conclude that the breakpoint in breathing frequency does not provide a valid method for the field-based assessment of the lactate or ventilatory thresholds in most subjects for running exercise.  相似文献   

20.
This study examined whether the ventilatory (V) compensation for metabolic acidosis with increasing O2 uptake (VO2) and CO2 output (VCO2) might be more in accord with the theoretical expectation of a progressive acceleration of proton production from carbohydrate oxidation rather than a sudden onset of blood lactate (BLa) accumulation. The interrelationships between V, VO2, VCO2 and BLa concentration, [BLa], were investigated in 10 endurance-trained male cyclists during incremental (120 +/- 15 W min-1) exercise tests to exhaustion. Regression analyses on the V, VCO2 and [BLa] vs VO2 data revealed that all were better fitted by continuous Y = A.exp.[B.VO2] + C rate laws than by threshold linear rate equations (P < 0.0001). Plots of V vs VCO2 and [BLa] were also non-linear. Ventilation increased as an exponential V = 27 +/- 4.exp.[0.37 +/- 0.03.VCO2] function of VCO2 and as a hyperbolic function of [BLa]. In opposition to the 'anaerobic (lactate) threshold' hypothesis, we suggest these data are more readily explained by a continuous development of acidosis, rather than a sudden onset of BLa accumulation, during progressive exercise.  相似文献   

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